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Yousri M. Barri

Researcher at University of Arkansas for Medical Sciences

Publications -  15
Citations -  1043

Yousri M. Barri is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Kidney disease & Transplantation. The author has an hindex of 11, co-authored 15 publications receiving 986 citations.

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Results of autologous stem cell transplant in multiple myeloma patients with renal failure.

TL;DR: The role of auto-SCT early in the disease course and benefits of tandem SCT require further evaluation in the setting of multiple myeloma patients with renal failure as discussed by the authors.
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Oxidant Mechanisms in Gentamicin Nephrotoxicity

TL;DR: There is evidence to suggest that studies may have broader implication in being relevant to other aminoglycosides including streptomycin and being applicable to other major toxicity of am inoglycoside such as ototoxicity.
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Carbamylated low-density lipoprotein induces death of endothelial cells: a link to atherosclerosis in patients with kidney disease.

TL;DR: The studies demonstrated that cLDL but not nLDL induced dose-dependent vascular cell injuries relevant to atherosclerosis, which included the proliferation of vascular smooth muscle cells and endothelial cell death, which suggest the potential role of carbamylated LDL in accelerated Atherosclerosis in patients with chronic renal disease and, possibly, in healthy individuals.
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Podocyte injury associated glomerulopathies induced by pamidronate.

TL;DR: Pamidronate has been mainly associated with collapsing focal segmental glomerulosclerosis and other glomerular diseases linked with podocyte injury and is needed to define the cause of the variability of renal histology with this agent.
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Polyoma viral infection in renal transplantation: the role of immunosuppressive therapy

TL;DR: Reduction in immunosuppression was associated with the stabilization of renal function when instituted early, and the interstitial nephritis associated with polyoma viral infection appears to be an inflammatory response to the virus rather than acute rejection.