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Showing papers by "Yuki Yoshida published in 2009"


Journal ArticleDOI
TL;DR: It is shown that mutations in either the biosynthetic or signaling pathway of jasmonates (JAs), long-distance wound signals, abolish the wound-induced formation of trichomes in Arabidopsis.
Abstract: Once attacked by herbivores, plants regenerate new leaves with increased trichome density as an inducible defense. Trichomes are specified from neighboring epidermal cells through local cell-cell interactions in the leaf primordia. However, the molecular mechanism of how herbivore-induced damage at older leaves remodels the pattern of trichome fate specification at newly forming leaves is largely unknown. In this study, we show that mutations in either the biosynthetic or signaling pathway of jasmonates (JAs), long-distance wound signals, abolish the wound-induced formation of trichomes. To identify the factors linking JA signaling to trichome fate specification, we isolated a novel class of mutants, unarmed (urm), which lack trichome induction but show otherwise normal responses to JAs. URM9 encodes an Importin beta family protein, and URM23 is identical to TRANSPARENT TESTA GLABRA1 (TTG1), the product of which interacts with the bHLH transcription factor GLABRA3 (GL3). Loss of either URM9 or URM23 disrupts the subnuclear localization of GL3, thus implicating GL3 in trichome induction. The expression of GL3 was enhanced by JA treatment prior to trichome initiation. Genetic analysis of multiple trichome mutants shows that GL3, in concert with the R2R3-Myb transcription factor GLABRA1 (GL1), promotes trichome fate in response to JA in a dosage-dependent manner. These results indicate that GL3 is a key transcription factor of wound-induced trichome formation acting downstream of JA signaling in Arabidopsis.

182 citations


Journal ArticleDOI
TL;DR: A widely linear receiver is proposed for OFDMA and SC-FDMA systems to cope with the interuser interference problem due to Tx IQIs and a novel subcarrier allocation scheme is proposed, which has high tolerance to such TxIQ distortion.
Abstract: One limiting issue in implementing high-speed wireless systems is the impairment associated with analog processing due to component imperfections. In uplink transmission of multiuser systems, a major source of such impairment is in-phase/quadrature-phase imbalance (IQI) introduced at multiple transmitters. In this paper, we deal with orthogonal-frequency-division multiple access (OFDMA) and single-carrier frequency-division multiple access (SC-FDMA) which have received attention in recent years as physical layer protocol in WiMAX and 3GPP Long Term Evolution (LTE) and analyze the effect of the transmitter (Tx) IQIs on OFDMA and SC-FDMA receivers. To cope with the interuser interference problem due to Tx IQIs, we propose a widely linear receiver for OFDMA and SC-FDMA systems and also propose a novel subcarrier allocation scheme, which has high tolerance to such Tx IQ distortion.

55 citations


Journal ArticleDOI
TL;DR: An icelike supramolecular structure was found in a self-assembled quasi 3-D crystalline hydrate in between the layers of CuII tris(pyridylthio)methyl hydroxide complexes as discussed by the authors.
Abstract: An icelike supramolecular structure is found in a self-assembled quasi 3-D crystalline hydrate in-between the layers of CuII tris(pyridylthio)methyl hydroxide complexes, which is comparable with co...

7 citations


Proceedings ArticleDOI
19 Apr 2009
TL;DR: This paper analyzes the effect of the transmitter (Tx) IQI on OFDMA receiver and proposes a novel subcarrier allocation scheme, which has high tolerance to such Tx IQ distortion.
Abstract: One limiting issue in implementing high-speed wireless systems is the impairment associated with analog processing due to component imperfections. In uplink transmission of multiuser systems, a major source of such impairment is IQ imbalance (IQI) introduced at multiple transmitters. In this paper, we analyze the effect of the transmitter (Tx) IQI on OFDMA receiver. To cope with the inter-user interference problem due to Tx IQI, we propose a novel subcarrier allocation scheme, which has high tolerance to such Tx IQ distortion.