scispace - formally typeset
Y

Yunjiao Wang

Researcher at Third Military Medical University

Publications -  21
Citations -  357

Yunjiao Wang is an academic researcher from Third Military Medical University. The author has contributed to research in topics: Tendon & Tendinopathy. The author has an hindex of 7, co-authored 16 publications receiving 155 citations.

Papers
More filters
Journal ArticleDOI

Aspirin inhibits inflammation and scar formation in the injury tendon healing through regulating JNK/STAT-3 signalling pathway.

TL;DR: Aspirin, as the classical representative of non‐steroidal anti-inflammatory drugs (NSAIDs) for its anti‐inflammatory and analgesic actions, has been commonly used in treating tendinopathy.
Journal ArticleDOI

Exosomes from tendon stem cells promote injury tendon healing through balancing synthesis and degradation of the tendon extracellular matrix.

TL;DR: It is found that TSCs injection and exosomes injection significantly decreased matrix metalloproteinases (MMP)‐3 expression, increased expression of tissue inhibitor of met alloproteinase‐3 (TIMP‐3) and Col‐1a1, and increased biomechanical properties of the ultimate stress and maximum loading.
Journal ArticleDOI

Exosomes Derived from Bone Marrow Stromal Cells (BMSCs) Enhance Tendon-Bone Healing by Regulating Macrophage Polarization

TL;DR: Evidence is provided that the local administration of BMSC-Exos promotes the formation of fibrocartilage by increasing M2 macrophage polarization in tendon-to-bone healing, leading to improved biomechanical properties.
Journal ArticleDOI

Interleukin-15 facilitates muscle regeneration through modulation of fibro/adipogenic progenitors.

TL;DR: The findings supported the potential role of IL-15 as a modulator on fate of FAPs in injured muscle and as a novel therapy for chronic muscle injury.
Journal ArticleDOI

Aspirin inhibits adipogenesis of tendon stem cells and lipids accumulation in rat injury tendon through regulating PTEN/PI3K/AKT signalling.

TL;DR: By down‐regulating PTEN/PI3K/AKT signalling, aspirin inhibited adipogenesis of TSCs and fatty infiltration in injury tendon, promoted biomechanical properties and decreased rupture risk of injury tendon.