Y
Yunman Li
Researcher at China Pharmaceutical University
Publications - 134
Citations - 3132
Yunman Li is an academic researcher from China Pharmaceutical University. The author has contributed to research in topics: Ischemia & Neuroprotection. The author has an hindex of 27, co-authored 125 publications receiving 2318 citations.
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Neuroprotection by the soy isoflavone, genistein, via inhibition of mitochondria-dependent apoptosis pathways and reactive oxygen induced-NF-κB activation in a cerebral ischemia mouse model.
TL;DR: It is suggested that genistein has a neuroprotective effect in transient focal ischemia, which may involve regulation of mitochondria-dependent apoptosis pathways and suppression of ROS-induced NF-κB activation.
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Enhancement of transdermal delivery of theophylline using microemulsion vehicle.
TL;DR: Results of studies showed that microemulsion system of theophylline might be promising vehicles for the transdermal delivery of thephylline.
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Anti-inflammatory effects of maslinic acid, a natural triterpene, in cultured cortical astrocytes via suppression of nuclear factor-kappa B.
TL;DR: Results suggest that maslinic acid can potentially reduce neuroinflammation by inhibiting NF-κB signal transducer pathway in cultured cortical astrocytes.
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Maslinic acid, a natural inhibitor of glycogen phosphorylase, reduces cerebral ischemic injury in hyperglycemic rats by GLT‐1 up‐regulation
TL;DR: It is demonstrated that, in addition to showing promising antidiabetic properties, MA has a direct beneficial effect in cerebral ischemic injury, which may be correlated with the promotion of glutamate clearance by NF‐κB‐mediated GLT‐1 up‐regulation.
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Berberine attenuates ischemia–reperfusion injury through inhibiting HMGB1 release and NF-κB nuclear translocation
Jun-rong Zhu,Jun-rong Zhu,Hai-dan Lu,Hai-dan Lu,Chao Guo,Weirong Fang,Hong-Dong Zhao,Junshan Zhou,Feng Wang,Yan-li Zhao,Yan-li Zhao,Yunman Li,Ying-Dong Zhang,Ying-Dong Zhang,Chang-qing Yang,Jianguo Sun +15 more
TL;DR: It is demonstrated that berberine protects the brain from ischemia–reperfusion injury and the mechanism may rely on its anti-inflammatory effects mediated by suppressing the activation of HMGB1/TLR4/NF-κB signaling.