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Zhenghao Ye

Researcher at Huazhong University of Science and Technology

Publications -  8
Citations -  42

Zhenghao Ye is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Ulcerative colitis & Medicine. The author has an hindex of 2, co-authored 4 publications receiving 11 citations.

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Journal ArticleDOI

Association of Clinical Phenotypes in Haploinsufficiency A20 (HA20) With Disrupted Domains of A20.

TL;DR: HA20 is characterized by early-onset and the most common symptoms of HA20 are recurrent oral ulcers, fever and gastrointestinal ulcers and the ZnF domain may be more closely related to musculoskeletal disorders.
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Questionnaire assessment helps the self-management of patients with inflammatory bowel disease during the outbreak of Coronavirus Disease 2019.

TL;DR: The epidemic of COVID-19 might have a certain impact on many aspects of Hubei IBD patients within four weeks of traffic control after the outbreak, and doctors could utilize the results from the questionnaire to guide patients’ self-management.
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Anti-High Mobility Group Box 1 Neutralizing-Antibody Ameliorates Dextran Sodium Sulfate Colitis in Mice.

TL;DR: The protective effect of this antibody was enhanced in TLR4-deficient mice in some aspects, indicating that both additional HMGB1-mediated as well as TLR 4-mediated inflammatory signaling pathways were involved in the induction of colitis by DSS.
Posted ContentDOI

Questionnaire assessment helps the self-management of patients with inflammatory bowel disease during the outbreak of Coronavirus Disease 2019

TL;DR: The outbreak of COVID-19 might not have a significant impact on most Hubei IBD patients within one month after the traffic control, and the patient's disease condition could be assessed by the questionnaires.
Journal ArticleDOI

Upregulation of antimicrobial peptide expression in slc26a3-/- mice with colonic dysbiosis and barrier defect

TL;DR: The dysbiotic microbiota, low mucosal pH, and mucus barrier defect in slc26a3−/− colon are accompanied by a stark upregulation of the expression of a panel of antimicrobial proteins, which may explain the low inflammatory burden in the gut of these mice.