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The beta-2 receptor mediated glycogenolytic responses to catecholamines in the dog

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This article is published in Archives internationales de pharmacodynamie et de thérapie.The article was published on 1968-12-01 and is currently open access. It has received 34 citations till now. The article focuses on the topics: Beta (finance) & Receptor.

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Book ChapterDOI

The Classification of Adrenoceptors (Adrenergic Receptors). An Evaluation from the Standpoint of Receptor Theory

TL;DR: A wide variety of tissues undergo a change of functional state on exposure to noradrenaline or adrenaline, and agents which produce responses in tissues by interacting with adrenoceptors are referred to, for convenience, as adrenergic agonists.
Journal ArticleDOI

Metabolic responses to selective β-adrenergic stimulation in man

TL;DR: The responses of plasma free fatty acid, glucose, lactate, insulin and growth hormone to intravenous administration of the predominantly β-2 stimulant, salbutamol, were studied and tentative proposals are made regarding the nature of the β-receptor subtypes responsible for mediating these effects.
Journal ArticleDOI

Exercise performance and beta-blockade.

TL;DR: Individuals receiving β-blockade medication show greater adaptive response to physical conditioning during treatment with β1-selective than non- selective blockade probably because of greater training intensity with the former therapy.
Journal ArticleDOI

Pharmacological blockade of a β(2)AR-β-arrestin-1 signaling cascade prevents the accumulation of DNA damage in a behavioral stress model.

TL;DR: Chronic restraint stress in mice recapitulates the effects of isoproterenol infusion to reduce p53 levels and results in the accumulation of DNA damage in the frontal cortex of the brain, suggesting that the β2AR-β-arrestin-1 signaling pathway may represent an attractive therapeutic target to prevent some of the negative consequences of stress in the treatment of stress-related disorders.
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