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These results are consistent with the hypothesis that leptin acts directly on neurons within the mNTS to reduce food intake through an interaction with GI signal processing.
This suggests that the responses of these neurons in the ventral forebrain are related to sensory-specific satiety, an important phenomenon which regulates food intake.
Our study provided evidence that the AP neurons received vagal input from the proximal stomach and suggested a potential role of the AP in the regulation of food intake.
Neurons in this region may also respond to the sight of food and to its texture.
Open accessJournal ArticleDOI
Jennifer Beshel, Yi Zhong 
73 Citations
Our analysis of the Drosophila brain extends the role of a small number of hunger-sensing neurons to include food-odor value representation.

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