scispace - formally typeset
Search or ask a question

How many kDa is the LIKE EARLY STARVATION (LESV)? 


Best insight from top research papers

The LIKE EARLY STARVATION 1 (LESV) protein mentioned in the research by Pfister et al. is not specifically stated to have a molecular weight in kDa. However, in a different context by Senoo, a 60-kDa heat shock protein (HSP) is discussed in relation to stress-induced pancreatitis in rats. This 60-kDa HSP is distinct from the LESV protein mentioned in the other study. Therefore, based on the available information from the provided contexts, the molecular weight of the LESV protein remains unspecified, while the 60-kDa HSP is associated with stress responses in rats.

Answers from top 5 papers

More filters
Papers (5)Insight
Not addressed in the paper.
Not addressed in the paper.
Not addressed in the paper.
LIKE EARLY STARVATION 1 (LESV) protein's molecular weight in kDa is not specified in the provided abstract and title.
Not addressed in the paper.

Related Questions

The fast-paced species more sensitive to starvation than slow-paced species?5 answersThe sensitivity to starvation between fast-paced and slow-paced species varies based on the specific ecological and physiological traits studied. Research on damselflies did not find a direct link between growth rate (representing pace-of-life) and physiological differentiation or sensitivity to stressors like UV exposure. In contrast, a study on grasshoppers showed that freezing speed affected midgut pH differently in fast-growing and slow-growing populations, indicating potential sensitivity differences to environmental stressors. Additionally, in clams, the energy cost of metabolism during starvation differed between species, with one species showing higher energy demand and greater reduction in body components, suggesting varying sensitivities to starvation based on pace-of-life traits. Therefore, the relationship between pace-of-life and sensitivity to starvation is complex and influenced by multiple factors across different species.
What's known about ac16 cells under serum starvation?4 answersAC16 cells, a transformed human cardiac cell line, have been studied under serum starvation conditions. It has been shown that AC16 cells cultured in low serum conditions and treated with low-dose doxorubicin, UV 254 nm, or H2O2 exposure for up to 48 hours exhibit reduced proliferation and markers of senescence. Molecular markers such as increased p21 and H2A.X variant histone levels have been observed in AC16 cells under all three treatment conditions. However, the up-regulation of another senescence marker, p16, was not observed. Additionally, AC16 cells have been found to exhibit changes in protein and phosphoprotein levels after serum starvation, with increased expression of proteins involved in the PTEN-PI-3 kinase-AKT, EGFR-MAPK-Stat, and transcription activator-polyamine signaling pathways. These findings suggest that serum starvation affects the proliferation and senescence of AC16 cells and alters their protein expression patterns.
How serum starvation affects doubling time?4 answersSerum starvation has varying effects on doubling time depending on the cell type and experimental conditions. In some cases, serum starvation can reduce the basal activity of cells, suggesting a longer doubling time. However, available experimental data do not entirely support this notion, as serum starvation can induce a swift and dynamic response with qualitative and quantitative differences across different cell types. The effect of serum starvation on doubling time may also be influenced by other factors such as the presence of specific growth factors or stress-relieving drugs. Additionally, the duration of serum starvation may play a role, as longer periods of starvation can lead to alterations in cell proliferation kinetics, including a decrease in the mitotic index and migration rate of differentiated cells. Overall, the impact of serum starvation on doubling time is complex and can vary depending on the specific experimental conditions and cell type being studied.
How many hours can a human survive without food?5 answersA human can survive without food for a limited amount of time. The exact duration varies depending on factors such as individual health, body composition, and environmental conditions. Generally, a person can survive for several weeks without food, but this timeline can be significantly reduced if the individual is already malnourished or has underlying health issues.
Is starvation good for behavioural studies in larval zebrafish?5 answersStarvation can be beneficial for behavioral studies in larval zebrafish. Starvation-induced hepatic steatosis in zebrafish larvae can be used as a model to understand the molecular mechanisms underlying abnormal liver metabolism and lipid accumulation. Starvation stress in adult female zebrafish has been shown to have significant effects on reproductive impairments in both adults and larvae, including delayed embryonic hatching, decreased larval body length, disrupted swimming ability, and reduced survival rate. Additionally, starvation and re-feeding experiments have revealed changes in the expression levels of neuropeptides involved in feeding behavior, such as increased expression of agrp and galanin and decreased expression of pomca. Furthermore, intermittent fasting has been found to enhance behavior and neurotransmitter levels in larval zebrafish, potentially improving memory maintenance and reducing anxiety. Overall, starvation can provide valuable insights into the effects of nutrient deprivation on behavior and physiology in larval zebrafish.
What is the effect of starvation on membrane vesicle release?2 answersStarvation has been found to have an effect on membrane vesicle release in various organisms. In Bacillus subtilis, membrane vesicles (MVs) are released under stress conditions such as starvation. This release of MVs is independent of the SOS response and is triggered by autolysis, which is mediated by host-encoded cell wall-lytic enzymes. The MVs released during starvation can serve as a protective mechanism for the cellular membrane, neutralizing the damaging effects of stressors such as surfactants. In rat intestinal epithelial cells, starvation has been shown to increase the surface area of microvilli (MV) and the fluidity of brush border membranes (BBM). These changes in MV and BBM properties contribute to an increase in d-glucose transport through the cells' membranes during starvation. Overall, starvation induces membrane vesicle release and alters membrane properties to facilitate nutrient uptake and protect against stress-induced damage.