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Showing papers on "Altitude Hypoxia published in 1995"


Journal Article
TL;DR: A comparison of the subjects' verbalizations during the three interviews showed that they experienced a strong crisis during the first period at altitude, which improved partially during the second period, and evidence for use of certain defense mechanisms was found 8-20 h after ascent; both dependent measures were adversely affected.
Abstract: We examined the effects of altitude hypoxia on the subject's cognitive functions and psychological state. Two assessment approaches were used, a cognitive-motor task and a structured interview. An experimental group of six subjects was tested three times: once under conditions of normoxia, once 8-20 h after a rapid climb to an altitude of 4383 m, and once 48-60 h after ascent to that altitude. A comparison of the experimental group's performance on the cognitive-motor task with that of a control group showed a difference on the second administration: the experimental group's performance did not improve, whereas the control group's performance did. This difference recovered on the third administration. A comparison of the subjects' verbalizations during the three interviews showed that they experienced a strong crisis during the first period at altitude, which improved partially during the second period. Evidence for use of certain defense mechanisms, e.g. self-concern, was found 8-20 h after ascent; both dependent measures were adversely affected. These measures recovered after 48-60 h. Language: en

19 citations


Journal ArticleDOI
TL;DR: Preadaptation of rats to altitude hypoxia results in reduced activation of lipid peroxidation during subsequent stress, inflammation, or both, as compared toHypoxiaunadapted animals, with the result that secondary changes in organs and tissues of adapted rats are much less pronounced and conditions are created for alleviating the acute inflammation and the stress reaction.
Abstract: Preadaptation of rats to altitude hypoxia results in reduced activation of lipid peroxidation during subsequent stress, inflammation, or both, as compared to hypoxiaunadapted animals, with the result that secondary changes in organs and tissues of adapted rats are much less pronounced and conditions are created for alleviating the acute inflammation and the stress reaction.