Showing papers on "Mitochondrial biogenesis published in 1983"
TL;DR: Electron microscopic and electrophoretic analyses carried out on mtDNA molecules at different vitellogenic stages show that the frequency of displacement loop (D-loop) forms is correlated with the intensity of mitochondrial biogenesis.
33 citations
01 Jan 1983
TL;DR: Very little is known about either the genes which code for mitochondrial proteins, nor the environmental, physiological and genetic factors which regulate mitochondrial biogenesis and coordinate it with development of the plant cell.
Abstract: Many of the major changes which occur during growth and differentiation of higher plants are associated with, or dependent upon, marked changes in mitochondrial number, structure and activity However, very little is known about either the genes which code for mitochondrial proteins, nor the environmental, physiological and genetic factors which regulate mitochondrial biogenesis and coordinate it with development of the plant cell
14 citations
TL;DR: Two-dimensional immunoelectrophoresis and rocket-electrophoreis were used to investigate changes in the pattern of protein antigens in the differentiation of maize root cells to reveal the appearance of two stage-specific antigen which were not organ-specific proteins.
1 citations
01 Jan 1983
TL;DR: One, or a combination of these effects, appears to be responsible for the known reduction in fetal size at doses of methylmercury below which overt teratogenic or maternal toxicity are observed.
Abstract: There are relatively few studies concerning either the ultrastructural or biochemical effects of toxic elements on subcellular organelles. Ultrastructural studies which show cellular vesiculation, mitochondrial damage and cellular necrosis have been reported for lead and arsenate. Ultrastructural/biochemical data are available for the effects of methylmercury on fetal organisms. These studies show cellular vesiculation of hepatocytes, decreased synthesis of protein and DNA associated with inhibition of mitochondrial biogenesis and respiratory function. One, or a combination of these effects, appears to be responsible for the known reduction in fetal size at doses of methylmercury below which overt teratogenic or maternal toxicity are observed.