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A. A. M. Wilde
Researcher at University of Amsterdam
Publications - 49
Citations - 568
A. A. M. Wilde is an academic researcher from University of Amsterdam. The author has contributed to research in topics: Internal medicine & Medicine. The author has an hindex of 11, co-authored 37 publications receiving 499 citations. Previous affiliations of A. A. M. Wilde include King Abdulaziz University.
Papers
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Journal ArticleDOI
Recurrent and founder mutations in the Netherlands—Phospholamban p.Arg14del mutation causes arrhythmogenic cardiomyopathy
van der Paul Zwaag,I. A. W. Van Rijsingen,R. de Ruiter,Eline A. Nannenberg,Judith A. Groeneweg,Jan G. Post,R. N. W. Hauer,van Isabelle Gelder,van den Maarten Berg,van der Pim Harst,A. A. M. Wilde,van Peter Tintelen +11 more
TL;DR: The p.Arg14del mutation in the PLN gene is the most frequently identified mutation in Dutch cardiomyopathy patients and is highly prevalent in the general population in the northern part of the Netherlands.
Journal ArticleDOI
Founder mutations in hypertrophic cardiomyopathy patients in the Netherlands.
Imke Christiaans,Eline A. Nannenberg,Dennis Dooijes,Rosalie J.E. Jongbloed,Michelle Michels,Pieter G. Postema,Danielle Majoor-Krakauer,A. van den Wijngaard,Marcel M.A.M. Mannens,van Peter Tintelen,van Irene Langen,A. A. M. Wilde +11 more
TL;DR: The genetics of HCM, the genotype-phenotype relation of Dutch founder MYBPC3 gene mutations, the prevalence and the geographic distribution of the Dutch founder mutations, and the consequences for genetic counselling and testing are described.
Journal ArticleDOI
Combined leadless pacemaker and subcutaneous implantable defibrillator therapy: feasibility, safety, and performance
Fleur V.Y. Tjong,T F Brouwer,Lonneke Smeding,Kirsten Kooiman,J. R. de Groot,David Ligon,R. Sanghera,M.J. Schalij,A. A. M. Wilde,Reinoud E. Knops +9 more
TL;DR: Combined LP and S-ICD therapy appears feasible in all animal experiments and in one human subject, and no interference in sensing and pacing during intrinsic and paced rhythm was noted in both animal and human subjects.
Journal ArticleDOI
Founder mutations in the Netherlands: SCN5a 1795insD, the first described arrhythmia overlap syndrome and one of the largest and best characterised families worldwide.
Pieter G. Postema,M.P. van den Berg,J. P. van Tintelen,F. van den Heuvel,Maik J. Grundeken,Nynke Hofman,W. van der Roest,Eline A. Nannenberg,Ingrid P.C. Krapels,Connie R. Bezzina,A. A. M. Wilde +10 more
TL;DR: A Dutch family carrying the SCN5a 1795insD mutation is reviewed, which is at present one of the largest and best-described families worldwide and it is about to obtain new insights into the phenotypic variability in this monogenic arrhythmia syndrome.
Journal ArticleDOI
ECG determinants in adult patients with chronic right ventricular pressure overload caused by congenital heart disease: relation with plasma neurohormones and MRI parameters.
J G J Neffke,I. I. Tulevski,E. E. van der Wall,A. A. M. Wilde,D. J. Van Veldhuisen,Ali Dodge-Khatami,B.J.M. Mulder +6 more
TL;DR: ECG parameters worsened gradually in asymptomatic or minimally symptomatic patients with chronic RV pressure overload, regardless of the nature of their congenital heart disease, and a significant positive correlation was found between QRS duration and RVEDV.