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Abdul Matin
Researcher at Stanford University
Publications - 104
Citations - 8165
Abdul Matin is an academic researcher from Stanford University. The author has contributed to research in topics: Chromate conversion coating & Escherichia coli. The author has an hindex of 46, co-authored 102 publications receiving 7692 citations. Previous affiliations of Abdul Matin include Norwegian University of Science and Technology & Max Planck Society.
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Starvation-induced cross protection against heat or H2O2 challenge in Escherichia coli.
TL;DR: Glucose- or nitrogen-starved cultures of Escherichia coli exhibited enhanced resistance to heat (57 degrees C) or H2O2 (15 mM) challenge, compared with their exponentially growing counterparts.
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The molecular basis of carbon-starvation-induced general resistance in Escherichia coli.
TL;DR: At the onset of starvation Escherichia coli undergoes a temporally ordered program of starvation gene expression involving 40–80 genes which some four hours later yields cells possessing an enhanced general resistance, which can be potentially useful for selective expression of desired genes in metabolically sluggish populations.
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Differential fates of biomolecules delivered to target cells via extracellular vesicles
Masamitsu Kanada,Michael Bachmann,Jonathan Hardy,Daniel Omar Frimannson,Laura L. Bronsart,Andrew Wang,Matthew D. Sylvester,Tobi L. Schmidt,Roger L. Kaspar,Manish J. Butte,Abdul Matin,Christopher H. Contag +11 more
TL;DR: It is observed that only microvesicles (MVs) can functionally transfer loaded reporter molecules to recipient cells, largely by delivering plasmid DNA, and that exosomes and MVs are structurally and functionally distinct.
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Genetic basis of starvation survival in nondifferentiating bacteria.
TL;DR: 1) Macromolecular degradation; 2) Escape from starvation; 3) Development of a more resistant cell state; and 4) escape from starvation.
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The putative sigma factor KatF has a central role in development of starvation-mediated general resistance in Escherichia coli.
TL;DR: The KatF mutant failed to develop starvation-mediated cross protection to osmotic, oxidative, and heat stresses, and it exhibited an altered pattern of protein synthesis at elevated temperature.