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Aihua Liu
Researcher at Capital Medical University
Publications - 133
Citations - 1775
Aihua Liu is an academic researcher from Capital Medical University. The author has contributed to research in topics: Aneurysm & Embolization. The author has an hindex of 19, co-authored 111 publications receiving 1280 citations. Previous affiliations of Aihua Liu include Chinese Academy of Sciences & University of Science and Technology of China.
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Journal ArticleDOI
Comparative transcriptomic profiling of a salt-tolerant wild tomato species and a salt-sensitive tomato cultivar.
TL;DR: The specific down-regulation in PI365967 of a putative high-affinity nitrate transporter, known as a repressor of lateral root initiation, may explain the better root growth of this genotype during salt stress.
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Comparison of Stent-Assisted Coiling vs Coiling Alone in 563 Intracranial Aneurysms: Safety and Efficacy at a High-Volume Center.
TL;DR: Compared with coiling alone, stent-assisted coiling may achieve lower recurrence rates, with comparable procedure-related complications and clinical outcomes in both ruptured and unruptured aneurysms.
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Transcutaneous vagus nerve stimulation for refractory epilepsy: a randomized controlled trial
Peijing Rong,Aihua Liu,Jianguo Zhang,Yuping Wang,Wei He,Anchao Yang,Liang Li,Hui Ben,Liping Li,Huanguang Liu,Peng Wu,Rupeng Liu,Yufeng Zhao,Jianliang Zhang,Feng Huang,Xia Li,Bing Zhu +16 more
TL;DR: The results show that this ta-VNS treatment can effectively reduce the frequency of seizures and improve the patient's quality of life and may be an effective treatment for refractory epilepsy.
Journal Article
An alternative therapy for drug-resistant epilepsy: transcutaneous auricular vagus nerve stimulation.
Peijing Rong,Aihua Liu,Jie Zhang,Yun-Zhen Wang,Anchao Yang,Liang Li,Hui Ben,Rupeng Liu,Wei He,Huanguang Liu,Feng Huang,Xiao Li,Wu P,Bing Zhu +13 more
TL;DR: Ta‐VNS can suppress epileptic seizures and is a safe, effective, economical, and widely applicable treatment option for drug‐resistant epilepsy.
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Neuroprotective effects of miR-27a against traumatic brain injury via suppressing FoxO3a-mediated neuronal autophagy.
TL;DR: Overexpression of miR-27a significantly attenuated neurological deficits and brain injury, especially suppressed autophagic activation after TBI, and it was identified that miR -27a directly targeted the FoxO3a 3'UTR region to reduced FoxO 3a protein expression.