Akira Terao

TL;DR: These results indicate that mice lacking cryptochromes are not simply a genetic model of circadian arrhythmicity in rodents and functionally implicate cryptochromaes in the homeostatic regulation of sleep.

TL;DR: It is indicated that acute activation of MCH neurons is sufficient, but not necessary, to trigger the transition from NREM to REM sleep and that M CH neurons also play a role in the initiation and maintenance of NREM sleep.

TL;DR: The widespread increase of heat shock protein family mRNAs in brain during sleep deprivation may be a neuroprotective response to prolonged wakefulness, whereas the relatively limited heat shockprotein family mRNA expression during recovery sleep may be related to the role of heatshock proteins in protein biogenesis and thus to the restorative function of sleep.

TL;DR: It is found that the molecular response to sleep deprivation and recovery sleep in the brain is highly conserved between these two mammalian species, at least in terms of expression of immediate early gene and heat shock protein family members.

TL;DR: It is found that melanin concentrating hormone–producing neurons (MCH neurons) in the hypothalamus actively contribute to forgetting in rapid eye movement (REM) sleep and are involved in active forgetting in the hippocampus.