A
Akiva Apelbaum
Researcher at University of California, Davis
Publications - 12
Citations - 852
Akiva Apelbaum is an academic researcher from University of California, Davis. The author has contributed to research in topics: Ethylene & Enzyme. The author has an hindex of 9, co-authored 12 publications receiving 830 citations.
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Journal ArticleDOI
Polyamines inhibit biosynthesis of ethylene in higher plant tissue and fruit protoplasts.
Akiva Apelbaum,Alan C. Burgoon,James D. Anderson,Morris Lieberman,Ruth Ben-Arie,Autar K. Mattoo +5 more
TL;DR: Polyamines, as well as putrescine, inhibited auxin-induced ethylene production and the conversion of methionine and 1-aminocyclopropane-1-carboxylic acid to ethylene.
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Biosynthesis of stress ethylene induced by water deficit.
Akiva Apelbaum,Shang Fa Yang +1 more
TL;DR: Cycloheximide, an inhibitor of protein synthesis, effectively blocked both ethylene production and ACC formation, suggesting that water stress induces de novo synthesis of ACC synthase, which is the rate-controlling enzyme in the pathway of ethylene biosynthesis.
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Some Characteristics of the System Converting 1-Aminocyclopropane-1-carboxylic Acid to Ethylene
TL;DR: The data presented support the possibility that the conversion of exogenous ACC to C(2)H(4) proceeds via the natural physiological pathway.
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Evidence for transglutaminase activity in plant tissue
Isaac Icekson,Akiva Apelbaum +1 more
TL;DR: An extract prepared from the apical meristematic region of etiolated pea seedlings was able to catalyze the incorporation of putrescine into trichloroacetic acid precipitable material and was found to be soluble and followed a typical Michaelis-Menten kinetics.
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Inhibition of the Conversion of 1-Aminocyclopropane-1-carboxylic Acid to Ethylene by Structural Analogs, Inhibitors of Electron Transfer, Uncouplers of Oxidative Phosphorylation, and Free Radical Scavengers.
TL;DR: Short-chain organic acids from acetic to butyric acid and alpha-aminoisobutyric acid inhibited C(2)H(4) production at 1 to 5 millimolar and lowered endogenous ACC levels, and these inhibitions, like that of CCA, were overcome with ACC.