Ala Muala

TL;DR: The acute and chronic effects of air pollution exposure on the cardiovascular system are reviewed and how these effects may explain the observed increases in cardiovascular morbidity and mortality are discussed.

TL;DR: Acute exposure to wood smoke as a model of exposure to biomass combustion is associated with an immediate increase in central arterial stiffness and a simultaneous reduction in heart rate variability.

TL;DR: Diesel exhaust inhalation disturbs normal vascular homeostasis with enhanced NO generation unable to compensate for excess consumption, and is suggested to be mediated through reduced NO bioavailability.

TL;DR: Short-term exposure to sooty PAH rich wood smoke did not induce an acute neutrophilic inflammation, a classic hallmark of air pollution exposure in humans, and in vitro data demonstrated that wood smoke particles generated under these incomplete combustion conditions induced cell death and DNA damage, with only minor inflammatory responses.

TL;DR: A cabin air inlet particle filter including an active charcoal component was highly effective in reducing both DE particulate and gaseous components, with reduced exhaust-induced symptoms in healthy volunteers, demonstrating the effectiveness of cabin filters to protect subjects travelling in vehicles from diesel exhaust emissions.