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Alain H. Altamirano-Espinoza

Researcher at CINVESTAV

Publications -  15
Citations -  135

Alain H. Altamirano-Espinoza is an academic researcher from CINVESTAV. The author has contributed to research in topics: Stimulation & Agonist. The author has an hindex of 8, co-authored 13 publications receiving 113 citations.

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Analysis of anandamide- and lysophosphatidylinositol-induced inhibition of the vasopressor responses produced by sympathetic stimulation or noradrenaline in pithed rats.

TL;DR: The sympatho-inhibition by anandamide is primarily mediated by cannabinoid CB1 and, minimally, by cannabidiol-sensitive receptors, while LPI-induced inhibition of both vasopressor responses seems to be mainly mediated by postjunctional cannabidsensitive GPR55 receptors.
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Olcegepant blocks neurogenic and non-neurogenic CGRPergic vasodepressor responses and facilitates noradrenergic vasopressor responses in pithed rats

TL;DR: This randomized study investigated in pithed rats the effect of acute i.v. treatment with olcegepant on the neurogenic and non‐neurogenic responses of CGRPergic vasodepressor responses; and the noradrenergic vasopressor responses.
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The role of dopamine D2, but not D3 or D4, receptor subtypes, in quinpirole-induced inhibition of the cardioaccelerator sympathetic outflow in pithed rats.

TL;DR: The present study was designed to identify pharmacologically the specific D2‐like receptor subtypes involved in this sympathoinhibition by quinpirole.
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Intrathecal dihydroergotamine inhibits capsaicin-induced vasodilatation in the canine external carotid circulation via GR127935- and rauwolscine-sensitive receptors.

TL;DR: Intrathecal dihydroergotamine seems to inhibit the external carotid vasodilatation to capsaicin by spinal activation of serotonin 5-HT(1B/1D) (probably 5- HT( 1B)) receptors and α(2) ( probably α( 2A/2C))-adrenoceptors.
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Role of pre-junctional CB1, but not CB2 , TRPV1 or GPR55 receptors in anandamide-induced inhibition of the vasodepressor sensory CGRPergic outflow in pithed rats.

TL;DR: The results suggest that anandamide‐induced inhibition of the vasodepressor sensory CGRPergic outflow is mainly mediated by pre‐junctional activation of CB1 receptors, with no pharmacological evidence for the role of CB2, TRPV1 or GPR55 receptors.