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Alan Collmer

Researcher at Cornell University

Publications -  172
Citations -  18990

Alan Collmer is an academic researcher from Cornell University. The author has contributed to research in topics: Pseudomonas syringae & Effector. The author has an hindex of 74, co-authored 171 publications receiving 18155 citations. Previous affiliations of Alan Collmer include University of Maryland, College Park & Washington State University.

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Book ChapterDOI

Pectic Enzyme Production and Bacterial Plant Pathogenicity

TL;DR: A mutant derivative of the soft-rot pathogen Erwinia chrysanthemi, containing site-directed mutations in genes encoding all of the known extracellular pectic enzymes but still able to macerate plant tissues, was found to produce several novel pectate lyases in planta.
Journal ArticleDOI

Inhibition of the hypersensitive response in tobacco by pectate lyase

TL;DR: Heat-stable products released by pectate lyase from isolated tobacco cell walls were similarly able to suppress the hypersensitive response in tobacco leaves, suggesting that the protection phenomenon may be mediated by galacturonic acid-containing oligosaccharides.
Journal ArticleDOI

Attachment, Chemotaxis, and Multiplication of Agrobacterium tumefaciens Biovar 1 and Biovar 3 on Grapevine and Pea.

TL;DR: Polygalacturonase production may affect attachment to grape roots and multiplication of A. tumefaciens bv.
Journal ArticleDOI

GABA (γ-Aminobutyric Acid) Uptake Via the GABA Permease GabP Represses Virulence Gene Expression in Pseudomonas syringae pv. tomato DC3000.

TL;DR: It is demonstrated that the GABA permease GabP provides the primary mechanism for GABA uptake by tomato DC3000 and that the gabP deletion mutant ΔgabP is insensitive to GABA-mediated repression of T3SS expression, which supports the hypothesis that GABA uptake via GabP acts to regulate T3 SS expression in planta.
Journal ArticleDOI

Elicitation of the hypersensitive response by Pseudomonas syringae

TL;DR: This review focuses on the interaction of Pseudomonas syringae with non-host plants as a model system for the bacterial induction of the HR and discusses the genetics of Pseudo-syringae pathogenicity and HR elicitation.