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Alex Widmer

Researcher at ETH Zurich

Publications -  180
Citations -  9678

Alex Widmer is an academic researcher from ETH Zurich. The author has contributed to research in topics: Population & Silene latifolia. The author has an hindex of 53, co-authored 169 publications receiving 8643 citations. Previous affiliations of Alex Widmer include Indiana University & École Polytechnique Fédérale de Lausanne.

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Evidence for Degeneration of the Y Chromosome in the Dioecious Plant Silene latifolia

TL;DR: Testing whether the S. latifolia Y chromosome is undergoing genetic degeneration by analyzing seven sex-linked genes detects signs of degeneration in most of the Y- linked gene sequences analyzed, similar to those of animal Y-linked and neo-Y chromosome genes.
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Evolution of postzygotic reproductive isolation in a guild of deceptive orchids.

TL;DR: Investigation of a group of food‐deceptive Mediterranean orchids from the genera Anacamptis, Neotinea, and Orchis reveals that intrinsic postzygotic isolation strongly contributes to maintaining species boundaries among Mediterranean food‐ deceptive orchid while establishing a prominent role for these reproductive barriers in the early stage of species isolation.
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Genetic structure of hybrid zones between Silene latifolia and Silene dioica (Caryophyllaceae): evidence for introgressive hybridization

TL;DR: Analyzing the structure of two natural hybrid zones between Silene latifolia and Silene dioica in the Swiss Alps revealed that morphology in this study system is useful for identifying hybrids, but not for detailed analysis of hybrid zone structure.
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Environmental heat and salt stress induce transgenerational phenotypic changes in Arabidopsis thaliana.

Léonie Suter, +1 more
- 09 Apr 2013 - 
TL;DR: It is found that exposure to heat stress in previous generations accelerated flowering under G4 control conditions in Sha-0, but heritable effects disappeared in G5 after two generations without stress exposure, indicating that stress can induce heritable, potentially adaptive phenotypic changes, probably through epigenetic mechanisms.