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Alja Videtic

Researcher at University of Ljubljana

Publications -  8
Citations -  544

Alja Videtic is an academic researcher from University of Ljubljana. The author has contributed to research in topics: Poison control & Population. The author has an hindex of 7, co-authored 8 publications receiving 508 citations. Previous affiliations of Alja Videtic include University of Primorska.

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Increased BDNF promoter methylation in the Wernicke area of suicide subjects.

TL;DR: BDNF promoter/exon IV is frequently hypermethylated in the Wernicke area of the postmortem brain of suicide subjects irrespective of genome-wide methylation levels, indicating that a gene-specific increase in DNA methylation could cause or contribute to the downregulation of BDNF expression in suicide subjects.
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TrkB gene expression and DNA methylation state in Wernicke area does not associate with suicidal behavior

TL;DR: The data show that no correlation exists between TrkB gene methylation and suicide in Wernicke area, confirming that expression and methylation state of suicide-related genes, even belonging to the same pathway, may be specific for brain area.
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Suicide, stress and serotonin receptor 1A promoter polymorphism -1019C>G in Slovenian suicide victims.

TL;DR: This study provides no evidence for the implication of HTR1A promoter polymorphism in suicide in general, but it suggests further studies that would take into account the interconnected network of suicide completion, genetic background and stress, beside other risk factors.
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Promoter and functional polymorphisms of HTR2C and suicide victims.

TL;DR: Haplotype analysis on female population showed marginal association of haplotype G‐C with suicide, which speaks for the plausible implication of the HTR2C in suicide susceptibility.
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Serotonin transporter gene promoter (5-HTTLPR) and intron 2 (VNTR) polymorphisms: a study on Slovenian population of suicide victims.

TL;DR: The study showed no association of serotonin transporter polymorphisms and suicide and was performed on a population with a very high suicide rate and the role of 5-HTTLPR polymorphisms may be different in other populations.