A
Allan B. Dietz
Researcher at Mayo Clinic
Publications - 207
Citations - 8246
Allan B. Dietz is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Mesenchymal stem cell & Immune system. The author has an hindex of 46, co-authored 193 publications receiving 6986 citations. Previous affiliations of Allan B. Dietz include Texas A&M University & University of Louisville.
Papers
More filters
Journal ArticleDOI
Immunosuppressive CD14+HLA-DRlow/− Monocytes in Prostate Cancer
Stanimir Vuk-Pavlović,Peggy A. Bulur,Yi Lin,Rui Qin,Carol L. Szumlanski,Xinghua Zhao,Allan B. Dietz +6 more
TL;DR: To determine if the levels of circulating myeloid‐derived suppressor cells increase with progression of prostate cancer (PCa); to determine if such cells could contribute to the relative inefficiency of PCa immunotherapy.
Journal ArticleDOI
Immunosuppressive CD14+HLA-DRlow/− monocytes in B-cell non-Hodgkin lymphoma
Yi Lin,Michael P. Gustafson,Peggy A. Bulur,Dennis A. Gastineau,Thomas E. Witzig,Allan B. Dietz +5 more
TL;DR: It is reported that CD14(+)HLA-DR(low/-) monocytes are a major and multifactorial contributor to systemic immunosuppression in NHL.
Journal ArticleDOI
Normal human monocytes exposed to glioma cells acquire myeloid-derived suppressor cell-like properties.
Jennifer C. Rodrigues,Guido C. Gonzalez,Lei Zhang,George M. Ibrahim,John Kelly,John Kelly,Michael P. Gustafson,Yi Lin,Allan B. Dietz,Peter A. Forsyth,V. Wee Yong,V. Wee Yong,Ian F. Parney +12 more
TL;DR: It is suggested that MDSC may be an important contributor to systemic immunosuppression and can be modeled in vitro by GCM.
Journal ArticleDOI
Platelet lysate consisting of a natural repair proteome supports human mesenchymal stem cell proliferation and chromosomal stability.
Ruben J. Crespo-Diaz,Atta Behfar,Greg W. Butler,Douglas J. Padley,Michael G. Sarr,Jozef Bartunek,Allan B. Dietz,Andre Terzic +7 more
TL;DR: Human platelet lysate adherent to good manufacturing practices (GMP-hPL) provided a nonzoonotic adjuvant that enhanced the capacity of BM-hMSC to proliferate and accelerates hMSC proliferation with no chromosomal aberrancy, through an innate repair paradigm.
Journal ArticleDOI
High efficiency adenovirus-mediated gene transfer to human dendritic cells.
TL;DR: With the aid of liposome-mediated infection, gene transfer into CD83+ dendritic cells was highly effective, resulting in more than 90% of the cells expressing the transgene.