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Allan L. Reiss

Researcher at Stanford University

Publications -  553
Citations -  64704

Allan L. Reiss is an academic researcher from Stanford University. The author has contributed to research in topics: Fragile X syndrome & Autism. The author has an hindex of 118, co-authored 529 publications receiving 59363 citations. Previous affiliations of Allan L. Reiss include Johns Hopkins University School of Medicine & University of California, San Diego.

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Integrated functional genomic analyses of Klinefelter and Turner syndromes reveal global network effects of altered X chromosome dosage

TL;DR: A comparative analysis of TS vs. KS regarding differences at the genomic network level measured in primary samples by analyzing gene expression, DNA methylation, and chromatin conformation indicates the existence of common molecular mechanisms for gene regulation in TS and KS that transmit the gene dosage changes to the transcriptome.
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Neuroanatomical phenotype of Klinefelter syndrome in childhood: a voxel-based morphometry study.

TL;DR: A voxel-based morphometry study of regional gray and white matter volumes in KS males offers new insight into the relationships among X-chromosome gene expression, neuroanatomy, and cognitive-behavioral functions impaired in KS, including language and attention.
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Androgen Treatment Effects on Motor Function, Cognition, and Behavior in Boys with Klinefelter Syndrome

TL;DR: This double‐blind, randomized trial demonstrates that 24 months of childhood low‐dose androgen treatment in boys with Klinefelter syndrome benefited 1 of 5 primary endpoints (visual‐motor function).
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Prenatal exposure to organophosphate pesticides and functional neuroimaging in adolescents living in proximity to pesticide application.

TL;DR: This first functional neuroimaging study of prenatal OP exposure suggests that pesticides may impact cortical brain activation, which could underlie previously reported OP-related associations with cognitive and behavioral function.
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Cytogenetic studies of males with schizophrenia: Screening for the fragile X chromosome and other chromosomal abnormalities

TL;DR: No chromosomal aberrations or folate-sensitive fragile sites were found in samples from these patients, and cytogenetic screening of populations of patients might provide clues for further in depth molecular studies.