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Ameeth Vedre
Researcher at Michigan State University
Publications - 8
Citations - 406
Ameeth Vedre is an academic researcher from Michigan State University. The author has contributed to research in topics: Crystallization & Cholesterol. The author has an hindex of 7, co-authored 7 publications receiving 357 citations.
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Journal ArticleDOI
Effect of cholesterol crystals on plaques and intima in arteries of patients with acute coronary and cerebrovascular syndromes.
TL;DR: By avoiding ethanol in tissue preparation, CCs perforating the intima were shown to be associated with PD, and crystal content was significantly associated with clinical events, suggesting that cholesterol crystallization may have a role in PD.
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Effect of statins on cholesterol crystallization and atherosclerotic plaque stabilization.
TL;DR: In conclusion, statins decreased ΔVE by altering cholesterol crystallization and blunting sharp-tipped crystal structure and dissolving cholesterol crystals in human arteries in vivo and in vitro, providing plaque stabilization.
Journal ArticleDOI
Physical factors that trigger cholesterol crystallization leading to plaque rupture
Ameeth Vedre,Dorothy Pathak,Martin A. Crimp,Chee Lum,Manoochehr Koochesfahani,George S. Abela +5 more
TL;DR: Physical factors influenced both volume and rate of cholesterol crystallization, suggesting that local factors may play an important role in triggering plaque rupture.
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Plaque Rupture and Thrombosis Are Reduced by Lowering Cholesterol Levels and Crystallization With Ezetimibe and Are Correlated With Fluorodeoxyglucose Positron Emission Tomography
Roshan Patel,Abed Janoudi,Ameeth Vedre,Kusai Aziz,Umesh Tamhane,Jack Rubinstein,Oliver G. Abela,Kevin Berger,George S. Abela +8 more
TL;DR: Lowering cholesterol levels with ezetimibe reduced plaque burden, crystallization, and inflammation, preventing plaque disruption and thrombosis.
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Hypercholesterolemia and myocardial function evaluated via tissue doppler imaging.
TL;DR: Cholesterol load in the serum and myocardium was significantly associated with decreased systolic and diastolic function by TDI and lipid lowering was protective.