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Andrea Coppadoro

Researcher at University of Milan

Publications -  45
Citations -  1342

Andrea Coppadoro is an academic researcher from University of Milan. The author has contributed to research in topics: Mechanical ventilation & Medicine. The author has an hindex of 16, co-authored 40 publications receiving 1000 citations. Previous affiliations of Andrea Coppadoro include Health Science University & University of Milano-Bicocca.

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Persisting high levels of plasma pentraxin 3 over the first days after severe sepsis and septic shock onset are associated with mortality

TL;DR: Persisting high levels of circulating PTX3 over the first days from sepsis onset may be associated with mortality, and this study is based on a prospective trial regarding the impact of glycemic control on coagulation in sepsi.
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Estimation of patient's inspiratory effort from the electrical activity of the diaphragm.

TL;DR: The derivation of the Pmusc/Eadi index from Eadi and airway pressure during an expiratory occlusion enables a continuous estimate of patient’s inspiratory effort, stable within each patient under different conditions of ventilator assistance.
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A clinical assessment of the Mucus Shaver, a device to keep the endotracheal tube free from secretions

TL;DR: The Mucus Shaver is a safe, feasible, and efficient device for endotracheal tube cleaning in the clinical setting and is helpful in preventing endotrachal tube colonization by potentially harmful microorganisms.
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Pentraxin 3 in acute respiratory distress syndrome: an early marker of severity.

TL;DR: The results presented here show that pentraxin 3 is elevated in acute lung injury and acute respiratory distress syndrome and that its levels correlate with parameters of lungs injury and systemic involvement.
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Topographic distribution of tidal ventilation in acute respiratory distress syndrome: effects of positive end-expiratory pressure and pressure support.

TL;DR: In patients with acute respiratory distress syndrome undergoing pressure support ventilation, higher positive end-expiratory pressure and lower support levels increase the fraction of tidal ventilation reaching dependent lung regions, yielding more homogeneous ventilation and, possibly, better ventilation/perfusion coupling.