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Andrea Marcantoni

Researcher at University of Turin

Publications -  64
Citations -  2111

Andrea Marcantoni is an academic researcher from University of Turin. The author has contributed to research in topics: Voltage-dependent calcium channel & Exocytosis. The author has an hindex of 26, co-authored 50 publications receiving 1859 citations. Previous affiliations of Andrea Marcantoni include University of Paris & University of Nice Sophia Antipolis.

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Cardiac effects of ghrelin and its endogenous derivatives des-octanoyl ghrelin and des-Gln14-ghrelin

TL;DR: In this paper, the effects of synthetic peptidyl GHS hexarelin (1 μM), or the natural ghrelin (50 nM), on the tension developed by guinea pig papillary muscle and on L-type Ca2+ current (ICa) of isolated ventricular cells were studied.
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cAMP-Binding Protein Epac Induces Cardiomyocyte Hypertrophy

TL;DR: It is shown that Epac activation leads to morphological changes and induces expression of cardiac hypertrophic markers and is associated with a Ca2+-dependent activation of the small GTPase, Rac, which activates a prohypertrophic signaling pathway.
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Loss of Cav1.3 Channels Reveals the Critical Role of L-Type and BK Channel Coupling in Pacemaking Mouse Adrenal Chromaffin Cells

TL;DR: It is found that the interpulse pacemaker current was always net inward and dominated by LTCCs, and the role of Cav1.3 channels is highlighted, as subthreshold pacemaker channels in MCCs and new interesting features about their role in the control of firing and catecholamine secretion at rest and during sustained stimulations matching acute stress are opened.
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Chronic hypoxia up-regulates α1H T-type channels and low-threshold catecholamine secretion in rat chromaffin cells

TL;DR: It is shown that RCCs exposed to chronic hypoxia for 12–18 h in 3% O2 express comparable densities of functional T‐type channels that depolarize the resting cells and contribute to low‐voltage exocytosis.
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Cav1.3 and BK Channels for Timing and Regulating Cell Firing

TL;DR: The most critical aspects of Cav1.3 channel gating and its coupling to large conductance BK channels recently discovered in spontaneously firing neurons and neuroendocrine cells are reviewed with the aim of furnishing a converging view of the role that these two channel types play in the regulation of cell excitability.