Doing Qualitative Research

http://www.cognitiveneuroscience.it/wp-content/uploads/2015/10/Rossini_CliNeuro_2015.pdf

Non-invasive electrical and magnetic stimulation of the brain, spinal cord, roots and peripheral nerves: Basic principles and procedures for routine clinical and research application: An updated report from an I.F.C.N. Committee

The Statistics of Canada

Drawing on information from four recent national and two state-level population-based surveys, the analyses suggest that there are more than 8 million adults in the US who are lesbian, gay, or bisexual, comprising 3.5% of the adult population. There are also nearly 700,000 transgender individuals in the US. In total, the study suggests that approximately 9 million Americans – roughly the population of New Jersey – identify as LGBT. Key findings from the study include among adults who identify as lesbian, gay, or bisexual, bisexuals comprise a slight majority (1.8% compared to 1.7% who identify as lesbian or gay); women are substantially more likely than men to identify as bisexual; estimates of those who report any lifetime same-sex sexual behavior and any same-sex sexual attraction are substantially higher than estimates of those who identify as lesbian, gay, or bisexual. An estimated 19 million Americans (8.2%) report that they have engaged in same-sex sexual behavior and nearly 25.6 million Americans (11%) acknowledge at least some same-sex sexual attraction.

/pdf/how-many-people-are-lesbian-gay-bisexual-and-transgender-584k83e3zr.pdf

How Many People are Lesbian, Gay, Bisexual and Transgender? - eScholarship

Meeting: International Seminar on Information and Communication Technology Statistics, Seoul, Republic of Korea, 19-21 July 2010

Ministry of Statistics and Programme Implementation

Medical schools are increasingly called to include social responsibility in their mandates. As such, they are focusing their attention on the social determinants of health (SDOH) as key drivers in the health of the patients and communities they serve. However, underlying this emphasis on the SDOH is the assumption that teaching medical students about the SDOH will lead future physicians to take action to help achieve health equity. There is little evidence to support this belief. In many ways, the current approach to the SDOH within medical education positions them as "facts to be known" rather than as "conditions to be challenged and changed." Educators talk about poverty but not oppression, race but not racism, sex but not sexism, and homosexuality but not homophobia. The current approach to the SDOH may constrain or even incapacitate the ability of medical education to achieve the very goals it lauds, and in fact perpetuate inequity. In this article, the authors explore how "critical consciousness" and a recentering of the SDOH around justice and inequity can be used to deepen collective understanding of power, privilege, and the inequities embedded in social relationships in order to foster an active commitment to social justice among medical trainees. Rather than calling for minor curricular modifications, the authors argue that major structural and cultural transformations within medical education need to occur to make educational institutions truly socially responsible.

Teaching the Social Determinants of Health: A Path to Equity or a Road to Nowhere?

As a result of increased interest in global health, more and more medical students and trainees from the ‘developed world’ are working and studying in the ‘developing world’. However, while opportunities to do this important work increase, there has been insufficient development of ethical guidelines for students. It is often assumed that ethics training in developed world situations is applicable to health experiences globally. However, fundamental differences in both clinical and research settings necessitate an alternative paradigm of analysis. This article is intended for teachers who are responsible for preparing students prior to such experiences. A review of major ethical issues is presented, how they pertain to students, and a framework is outlined to help guide students in their work.

https://drexel.edu/~/media/Files/dornsife/GlobalHealthEthicsforStudents.ashx?la=en

Global health ethics for students

https://tspace.library.utoronto.ca/bitstream/1807/75939/1/Looking%20Beyond%20Income.pdf

Looking Beyond Income and Education: Socioeconomic Status Gradients Among Future High-Cost Users of Health Care.

Conditioning magnetic stimulation of the cerebellum suppresses the motor cortex 5–8 ms later, probably through activation of cerebellar Purkinje cells, which inhibit the dentatothalamocortical pathway. To further characterize this pathway, we examined several factors that may modulate its excitability. We tested the effects of different test motor evoked potential (MEP) amplitudes, voluntary activation of the target muscle, and arm extension that required activation of proximal arm muscles while maintaining relaxation of hand muscles. Surface electromyography was recorded from the right first dorsal interosseous (FDI) muscle. A double-cone coil centered 3 cm lateral to the inion was used for right cerebellar stimulation. The stimulus intensity was set at 5% below the threshold for activation of the corticospinal tract. A figure-of-eight coil was used for left motor cortex stimulation. Interstimulus intervals (ISIs) between 3 and 15 ms were studied. Small test MEPs of about 0.5 mV were markedly inhibited at ISIs of 5–8 ms, but there was much less inhibition for test MEPs of about 2 mV. There was no significant MEP suppression during voluntary activation of the FDI muscle or during right arm extension. Left arm extension did not affect inhibition. Our findings indicate that cerebellar stimulation has a much stronger effect on motor cortex neurons activated near threshold intensities than those activated at higher intensities. Activation of contralateral but not ipsilateral proximal arm muscles during arm extension reduced the excitability of the cerebellothalamocortical projections to the hand area of the motor cortex.

Suppression of the motor cortex by magnetic stimulation of the cerebellum.

Objective

Lesioning or stimulation of the cerebellar thalamus is an established treatment for rest and postural tremors in Parkinson disease (PD). The cerebellothalamocortical (CTC) pathway can be assessed by transcranial magnetic stimulation (TMS) of the cerebellum, which suppresses the contralateral primary motor cortex (M1), a phenomenon termed cerebellar inhibition (CBI). Tremor reset can be used to assess whether the stimulated brain area is involved in the generation or transmission of tremor. We tested whether M1 or cerebellar stimulation can reset PD tremor, and investigated the excitability of the CTC pathway in PD.



Methods

Ten mild to moderate PD patients in the OFF medication state and 10 healthy controls were studied. Tremor reset was tested with TMS delivered to the cerebellum or M1. CBI was assessed by cerebellar stimulation followed by M1 stimulation at interstimulus intervals of 3 to 8 milliseconds. Subjects were tested both at rest and during arm extension.



Results

Rest tremor in PD was reset by M1 stimulation but not by cerebellar stimulation. Postural tremor was reset by both types of stimulation. At rest, CBI was reduced in PD patients compared to controls. Arm extension decreased CBI in controls and turned the inhibition into facilitation in patients. CBI correlated with the degree of tremor reset caused by the cerebellar stimulation.



Interpretation

The excitability of CTC pathway is decreased in PD. Rest and postural tremors in PD are mediated by different neuronal pathways, and the CTC pathway is involved in the generation or transmission of postural tremor. ANN NEUROL 2010

Andrew D. Pinto

Papers

Teaching the Social Determinants of Health: A Path to Equity or a Road to Nowhere?

Global health ethics for students

Looking Beyond Income and Education: Socioeconomic Status Gradients Among Future High-Cost Users of Health Care.

Suppression of the motor cortex by magnetic stimulation of the cerebellum.

Involvement of the Cerebellothalamocortical Pathway in Parkinson Disease