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Andrew St John
Researcher at Australian Research Council
Publications - 52
Citations - 1266
Andrew St John is an academic researcher from Australian Research Council. The author has contributed to research in topics: Cost effectiveness & Health care. The author has an hindex of 18, co-authored 49 publications receiving 1138 citations. Previous affiliations of Andrew St John include Royal Perth Hospital & Bond University.
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Ion Selective Electrodes (ISEs) and interferences--a review.
TL;DR: Ion Selective Electrodes are used to measure some of the most critical analytes on clinical laboratory and point-of-care analysers and they are not free of interferences.
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The use of urinary dipstick tests to exclude urinary tract infection: a systematic review of the literature.
TL;DR: The leukocyte esterase or nitrite test combination, with one or the other test positive, was used in 14 studies, and showed the highest and the lowest negative likelihood ratio.
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The effects of menopause and age on calcitropic hormones: A cross-sectional study of 655 healthy women aged 35 to 90
Richard L. Prince,Richard L. Prince,Ian M. Dick,Amanda Devine,Roger I. Price,Donald H. Gutteridge,Deborah A. Kerr,Arthur Criddle,Peter Garcia-Webb,Andrew St John +9 more
TL;DR: In this paper, the authors examined calcium-related metabolic factors in 655 healthy women and found that after menopause there is an increase in bone turnover and increased urine calcium loss independent of any effect of PTH or calcitriol.
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The value of self-monitoring of blood glucose: a review of recent evidence.
TL;DR: The present meta-analysis showed an SMBG-related HbA(1c) reduction in non-insulin-treated type 2 diabetes patients that was similar to that in previous systematic reviews but in a substantially larger patient sample.
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Determinants of intact parathyroid hormone and free 1,25-dihydroxyvitamin D levels in mild and moderate renal failure.
Andrew St John,Mark B. Thomas,Charmian P. Davies,Brian Mullan,Ian M. Dick,Brian G. Hutchison,Agatha van der Schaff,Richard L. Prince +7 more
TL;DR: The secondary hyperparathyroidism which occurs very early in the onset of chronic renal failure may be due to a reduction in the circulating concentration of 1,25(OH)2D consequent upon the renal failure.