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Andrew T. Meram

Researcher at LSU Health Sciences Center Shreveport

Publications -  16
Citations -  129

Andrew T. Meram is an academic researcher from LSU Health Sciences Center Shreveport. The author has contributed to research in topics: Medicine & DNA damage. The author has an hindex of 5, co-authored 14 publications receiving 64 citations.

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Hydrogen Sulfide and Hydrogen Sulfide-Synthesizing Enzymes Are Altered in a Case of Oral Adenoid Cystic Carcinoma.

TL;DR: This work reports a case of oral cavity ACC in a 54-year-old woman, and for the second time H2S concentrations have been directly quantified inside a human tumor, indicating that alterations in H 2S synthesis and metabolism may be important in the pathobiology of ACC.
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Is the Three-Dimensional Strut Plate an Adequate Fixation Technique for Mandibular Symphysis Fractures?

TL;DR: A 3-dimensional strut plate applied to symphysis fractures provides adequate fracture stabilization with a risk of complications comparable to that of more traditional fixation methods, and has the added advantage of minimal manipulation and adaptation.
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Nicotinamide Phosphoribosyl Transferase Is Increased in Osteosarcomas and Chondrosarcomas Compared to Benign Bone and Cartilage.

TL;DR: The data indicate that NAMPT plays a role in bone sarcomas and GCTs, and its higher expression may contribute to increased tumor aggressiveness.
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Porcine Urinary Bladder Extracellular Matrix for the Salvage of Fibula Free Flap Skin Paddle: Technical Note and Description of a Case:

TL;DR: A novel application of a commercially available porcine urinary bladder extracellular matrix, MatriStem, is established in the salvage of partial loss of the skin paddle of a fibula osteoseptocutaneous free flap that was utilized for mandibular reconstruction.
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Hydrogen Sulfide-Synthesizing Enzymes Are Altered in a Case of Oral Cavity Mucoepidermoid Carcinoma.

TL;DR: The results replicate those of two previous studies where the H2S-synthesizing enzymes are increased in a malignant tumor, while free H2H2S is either not increased or only slightly increased, suggesting that malignant tumors rapidly metabolize H1S as part of tumor maintenance and growth.