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Angelo De Stradis

Researcher at National Research Council

Publications -  49
Citations -  1445

Angelo De Stradis is an academic researcher from National Research Council. The author has contributed to research in topics: Biology & Xylella fastidiosa. The author has an hindex of 20, co-authored 38 publications receiving 1168 citations. Previous affiliations of Angelo De Stradis include University of Bari.

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Infectivity and Transmission of Xylella fastidiosa by Philaenus spumarius (Hemiptera: Aphrophoridae) in Apulia, Italy

TL;DR: The discovery of Xylella fastidiosa from olive trees with “Olive quick decline syndrome” in October 2013 on the west coast of the Salento Peninsula prompted an immediate search for insect vectors of the bacterium, and transmission tests showed P. spumarius as a vector of X. fastidioa strain infecting olives trees in theSalento Peninsula, Italy.
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Spittlebugs as vectors of Xylella fastidiosa in olive orchards in Italy

TL;DR: The biology and transmission ability of the meadow spittlebug, Philaenus spumarius, which was recently demonstrated to transmit X. fastidiosa to periwinkle plants, is investigated to plan an effective IPM strategy and limit further spread of the fastidious bacterium.
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A multipartite single-stranded negative-sense RNA virus is the putative agent of fig mosaic disease.

TL;DR: The consistent association of D MBs with mosaic symptoms and the results of molecular investigations strongly indicate that DMBs are particles of FMV, the aetiological agent of fig mosaic disease.
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High-level expression of the HIV-1 Pr55gag polyprotein in transgenic tobacco chloroplasts

TL;DR: Plastid transformation is a promising tool for HIV antigen manufacturing in plant cells by integrating and expression of the gag transgene in the plastome resulted in significantly higher protein accumulation levels.
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A Viroid RNA with a Specific Structural Motif Inhibits Chloroplast Development

TL;DR: RNA gel blot and in situ hybridizations revealed that PLMVd replicates in the albino leaf sectors and that it can invade the shoot apical meristem and induce alterations in proplastids, bypassing the RNA surveillance system that restricts the entry of a nucleus-replicating viroid and most RNA viruses.