Anja Hagen

TL;DR: Antiarrhythmic peptides are effective against ventricular tachyarrhythmias, such as late ischaemic ventricular fibrillation, CaCl2 or aconitine-induced arrhythmia, and it is still a matter of debate whether these drugs also act against atrialfibrillation.

TL;DR: The present review elucidates the implications of passive electrical properties for cardiac rhythm and arrhythmogenesis, and suggests a balanced geometrical distribution of gap junctions and reduced gap junction conductance may allow successful propagation of electrical impulse propagation.

TL;DR: The antiarrhythmic peptide AAP10, which improves gap-junctional intercellular coupling and prevents uncoupling by acidification in human cardiomyocytes, might be useful for antiarrHythmic strategies regarding arrhythmias caused by uncouplings of Cx43 and Cx45, but not Cx40.

TL;DR: Reduced intercellular endothelial communication together with programmed cell death helps to explain the toxic effect of nicotine leading to endothelial dysfunction.

TL;DR: The patient, a 19‐year‐old woman, has a multiple congenital anomaly syndrome with severe developmental and growth delay, muscular hypotonia, iris coloboma, abnormal external ears, widely spaced nipples, atrial septum defect, clubfoot, and arthrogryposis multiplex congenita.