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Ankur B. Dalia
Researcher at Indiana University
Publications - 79
Citations - 2391
Ankur B. Dalia is an academic researcher from Indiana University. The author has contributed to research in topics: Vibrio cholerae & Pilus. The author has an hindex of 24, co-authored 68 publications receiving 1799 citations. Previous affiliations of Ankur B. Dalia include University of Pennsylvania & Howard Hughes Medical Institute.
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Journal ArticleDOI
Retraction of DNA-bound type IV competence pili initiates DNA uptake during natural transformation in Vibrio cholerae.
Courtney K. Ellison,Triana N. Dalia,Alfredo Vidal Ceballos,Alfredo Vidal Ceballos,Joseph Che Yen Wang,Nicolas Biais,Nicolas Biais,Yves V. Brun,Ankur B. Dalia +8 more
TL;DR: Results indicate that type IV competence pili directly bind to DNA via their tip and mediate DNA internalization through retraction during this conserved mechanism of horizontal gene transfer.
Journal ArticleDOI
Obstruction of pilus retraction stimulates bacterial surface sensing
Courtney K. Ellison,Jingbo Kan,Jingbo Kan,Rebecca S. Dillard,David T. Kysela,Adrien Ducret,Cécile Berne,Cheri M. Hampton,Zunlong Ke,Zunlong Ke,Elizabeth R. Wright,Nicolas Biais,Nicolas Biais,Ankur B. Dalia,Yves V. Brun +14 more
TL;DR: To sense surfaces, bacteria use the resistance on retracting, surface-bound pili that occurs upon surface contact, which is critical for bacteria to recognize surface contact and initiate physiological changes required for surface-associated lifestyles.
Journal ArticleDOI
Multiplex genome editing by natural transformation.
TL;DR: MuGENT is a broadly applicable platform for accelerated evolution and genetic interaction studies in diverse naturally competent species and is found that natural cotransformation is an effective method for multiplex genome editing.
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Three surface exoglycosidases from Streptococcus pneumoniae, NanA, BgaA, and StrH, promote resistance to opsonophagocytic killing by human neutrophils
TL;DR: It is demonstrated that deglycosylation of a human serum glycoconjugate by the combined effects of NanA, BgaA, and StrH, is important for resistance to complement deposition and subsequent phagocytic killing of S. pneumoniae.
Journal ArticleDOI
Minimization of Bacterial Size Allows for Complement Evasion and Is Overcome by the Agglutinating Effect of Antibody
Ankur B. Dalia,Jeffrey N. Weiser +1 more
TL;DR: It is shown that in Streptococcus pneumoniae, increasing bacterial chain length sensitizes this pathogen to complement deposition and subsequent uptake by human neutrophils, and that minimizing chain length provides wild-type bacteria with a competitive advantage in vivo in a model of systemic infection.