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Andrew Camilli
Researcher at Tufts University
Publications - 177
Citations - 17943
Andrew Camilli is an academic researcher from Tufts University. The author has contributed to research in topics: Vibrio cholerae & Gene. The author has an hindex of 71, co-authored 166 publications receiving 15886 citations. Previous affiliations of Andrew Camilli include University of Maryland, Baltimore & Howard Hughes Medical Institute.
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Journal ArticleDOI
Bacterial small-molecule signaling pathways.
TL;DR: Recent research into two fundamental bacterial small-molecule signaling pathways are reviewed, suggesting how these two pathways may converge to control complex processes including multicellularity, biofilm formation, and virulence.
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Tn-seq: high-throughput parallel sequencing for fitness and genetic interaction studies in microorganisms.
TL;DR: Fitness for each gene of Streptococcus pneumoniae, a causative agent of pneumonia and meningitis is determined using a method based on the assembly of a saturated Mariner transposon insertion library.
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Definitions and guidelines for research on antibiotic persistence
Naomi N.Q. Balaban,Sophie Helaine,Kim Lewis,Martin Ackermann,Martin Ackermann,Bree B. Aldridge,Dan I. Andersson,Mark P. Brynildsen,Dirk Bumann,Andrew Camilli,James J. Collins,James J. Collins,James J. Collins,Christoph Dehio,Sarah M. Fortune,Jean-Marc Ghigo,Wolf-Dietrich Hardt,Alexander Harms,Matthias Heinemann,Deborah T. Hung,Urs Jenal,Bruce R. Levin,Jan Michiels,Gisela Storz,Man-Wah Tan,Tanel Tenson,Laurence Van Melderen,Annelies S. Zinkernagel +27 more
TL;DR: Scientists working on the response of bacteria to antibiotics define antibiotic persistence and provide practical guidance on how to study bacterial persister cells, and provide a guide to measuring persistence.
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Large-scale identification of serotype 4 Streptococcus pneumoniae virulence factors.
David L. Hava,Andrew Camilli +1 more
TL;DR: Four classes of mutants defective in infection models of the lung, lung and blood, and nasopharynx were identified, thus demonstrating the ex‐istence of tissue‐specific pneumococcal virulence factors.
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Cholera transmission: the host, pathogen and bacteriophage dynamic
TL;DR: Advances that will help to unravel how interactions between the host, the bacterial pathogen and the lytic bacteriophage might propel and quench cholera outbreaks in endemic settings and in emergent epidemic regions such as Zimbabwe are highlighted.