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Anne Bol

Researcher at Université catholique de Louvain

Publications -  144
Citations -  6430

Anne Bol is an academic researcher from Université catholique de Louvain. The author has contributed to research in topics: Positron emission tomography & Neutron. The author has an hindex of 38, co-authored 144 publications receiving 6172 citations. Previous affiliations of Anne Bol include Cliniques Universitaires Saint-Luc & Catholic University of Leuven.

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Mechanisms of chronic regional postischemic dysfunction in humans. New insights from the study of noninfarcted collateral-dependent myocardium.

TL;DR: A subgroup of patients with noninfarcted collateral-dependent myocardium, immature or insufficiently developed collaterals do not provide adequate flow reserve and it is proposed that these alterations result from repeated episodes of ischemia as opposed to chronic hypoperfusion.
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Coronary flow reserve calculated from pressure measurements in humans. Validation with positron emission tomography.

TL;DR: Fractional flow Reserve derived from pressure measurements correlates more closely to relative flow reserve derived from PET than angiographic parameters, validates in humans the use of fractional flow reserve as an index of the physiological consequences of a given coronary artery stenosis.
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A gradient-based method for segmenting FDG-PET images: methodology and validation

TL;DR: The gradient-based segmentation method applied on denoised and deblurred FDG-PET images proved to be more accurate than the source-to-background ratio method.
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Tri-dimensional automatic segmentation of PET volumes based on measured source-to-background ratios: influence of reconstruction algorithms.

TL;DR: A method for automatic volume segmentation of functional imaging based on a relationship between source-to-background ratio and the iso-activity level to be used is described, which has been established with radioactive spheres in a phantom.
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Glucose utilization in human visual cortex is abnormally elevated in blindness of early onset but decreased in blindness of late onset.

TL;DR: This unexpected difference between early and late blind subjects might reflect the persistence, in early blindness, of supranumerary synapses which would escape the normal developmental decrease in synaptic density during infancy.