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Annette Schröder
Researcher at University of Mainz
Publications - 63
Citations - 1704
Annette Schröder is an academic researcher from University of Mainz. The author has contributed to research in topics: Urinary bladder & Bladder outlet obstruction. The author has an hindex of 19, co-authored 60 publications receiving 1567 citations. Previous affiliations of Annette Schröder include Albany College of Pharmacy and Health Sciences & Karolinska Institutet.
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Journal ArticleDOI
EAU Guidelines on Urinary Incontinence
Joachim W. Thüroff,Paul Abrams,Karl-Erik Andersson,Walter Artibani,Christopher R. Chapple,Marcus J. Drake,Christian Hampel,Andreas Neisius,Annette Schröder,Andrea Tubaro +9 more
TL;DR: A summary of the 2009 update of the EAU guidelines on urinary incontinence, which provides ratings of the evidence and graded recommendations for the appropriate assessment and according treatment options and put them into clinical perspective is presented.
Journal ArticleDOI
EAU guidelines on urinary incontinence
Joachim W. Thüroff,Paul Abrams,Karl-Erik Andersson,Walter Artibani,Christopher R. Chapple,Marcus J. Drake,Christian Hampel,Andreas Neisius,Annette Schröder,Andrea Tubaro +9 more
TL;DR: A summary of the 2009 update of the EAU guidelines on urinary incontinence is presented, which provides ratings of the evidence and graded recommendations for the appropriate assessment and according treatment options and put them into clinical perspective.
Journal ArticleDOI
Effect of chronic bladder outlet obstruction on blood flow of the rabbit bladder
Annette Schröder,Paul Chichester,Barry A. Kogan,Penelope A. Longhurst,Jeremy Lieb,Anurag K. Das,Robert M. Levin +6 more
TL;DR: It is hypothesized that decreased blood flow to bladder smooth muscle is an etiological factor in bladder contractile dysfunction (bladder decompensation) secondary to partial outlet obstruction.
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Detrusor responses to prostaglandin E2 and bladder outlet obstruction in wild-type and EP1 receptor knockout mice
TL;DR: The EP1 receptor appears not to be essential for normal micturition or the mediation of bladder hypertrophy due to BOO but it seems to have a role in the development of detrusor overactivity caused by PGE2 and outlet obstruction.
Journal ArticleDOI
Bladder augmentation and urinary diversion in patients with neurogenic bladder: Surgical considerations
TL;DR: To prevent metabolic complications, careful patient selection and meticulous and lifelong follow up, as well as prophylactic treatment, are mandatory.