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Antonio L'Abbate

Researcher at Sant'Anna School of Advanced Studies

Publications -  578
Citations -  18384

Antonio L'Abbate is an academic researcher from Sant'Anna School of Advanced Studies. The author has contributed to research in topics: Myocardial infarction & Medicine. The author has an hindex of 70, co-authored 494 publications receiving 17587 citations. Previous affiliations of Antonio L'Abbate include University of Pisa & Marche Polytechnic University.

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“Variant” angina: One aspect of a continuous spectrum of vasospastic myocardial ischemia: Pathogenetic Mechanisms, Estimated Incidence and Clinical and Coronary Arteriographic Findings in 138 Patients

TL;DR: Angiography in all 37 patients studied during angina revealed a severe coronary vasospasm involving vessels with extremely variable extent of atherosclerosis, which may evolve into acute myocardial infarction and sudden death.
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Coronary vasospasm as a possible cause of myocardial infarction. A conclusion derived from the study of "preinfarction" angina.

TL;DR: In all 76 patients who underwent hemodynamic monitoring, 201thallium myocardial scintigraphy or angiography during angina, a vasospastic origin of the attacks was documented, and after infarction, complete thrombotic occlusion of the branch shown to undergo vasospasm was documented in two patients by angiographic.
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Stress echocardiography and the human factor: The importance of being expert

TL;DR: One hundred stress echocardiographic studies are more than adequate to build the individual learning curve and reach the plateau of diagnostic accuracy that the test can yield.
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Low T3 Syndrome. A strong prognostic predictor of death in patients with heart disease

TL;DR: Low-T3 syndrome is a strong predictor of death in cardiac patients and might be directly implicated in the poor prognosis of cardiac patients.
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Prognostic Role of Myocardial Blood Flow Impairment in Idiopathic Left Ventricular Dysfunction

TL;DR: It is demonstrated that severely depressed MBF is a predictor of poor prognosis in patients with idiopathic LV dysfunction independently of the degree of LV functional impairment and of the presence of overt heart failure.