F
Fabio Lattanzi
Researcher at University of Pisa
Publications - 95
Citations - 4079
Fabio Lattanzi is an academic researcher from University of Pisa. The author has contributed to research in topics: Dipyridamole & Coronary artery disease. The author has an hindex of 33, co-authored 93 publications receiving 3964 citations.
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Journal ArticleDOI
Stress echocardiography and the human factor: The importance of being expert
TL;DR: One hundred stress echocardiographic studies are more than adequate to build the individual learning curve and reach the plateau of diagnostic accuracy that the test can yield.
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High dose dipyridamole echocardiography test in effort angina pectoris
TL;DR: The dipyridamole echocardiography test was performed in 93 patients with effort chest pain and in 10 control subjects and had an overall specificity higher than that of the exercise stress test, and a similar overall sensitivity.
Journal ArticleDOI
In vivo quantitative ultrasonic evaluation of myocardial fibrosis in humans.
Eugenio Picano,Gualtiero Pelosi,Mario Marzilli,Fabio Lattanzi,Antonio Benassi,Luigi Landini,Antonio L'Abbate +6 more
TL;DR: In vivo on-line quantitative ultrasound analysis is feasible in man and reliably identifies variations in the regional extent of fibrosis in human myocardium as estimated by quantitative histology of endomyocardial biopsies.
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Dipyridamole-echocardiography test in effort angina pectoris
Eugenio Picano,Alessandro Distante,Michele Masini,Maria Aurora Morales,Fabio Lattanzi,Antonio L'Abbate +5 more
TL;DR: The dipyridamole-echocardiography test has a lower overall sensitivity in detecting CAD than EST but a higher specificity, detects the site of apparent ischemia as identified by regional asynergy more precisely than EST, and can unmask electrocardiographically silent effort ischemIA.
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Prognostic importance of dipyridamole-echocardiography test in coronary artery disease.
Eugenio Picano,Silva Severi,Claudio Michelassi,Fabio Lattanzi,Michele Masini,Enrico Orsini,Alessandro Distante,Antonio L'Abbate +7 more
TL;DR: It is concluded that the presence and timing of a transient dyssynergy during dipyridamole stress are useful predictors of subsequent cardiac events.