A
Arduino A. Mangoni
Researcher at Flinders Medical Centre
Publications - 397
Citations - 10350
Arduino A. Mangoni is an academic researcher from Flinders Medical Centre. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 42, co-authored 348 publications receiving 7835 citations. Previous affiliations of Arduino A. Mangoni include University of Cambridge & King's College London.
Papers
More filters
Journal ArticleDOI
Meta-analysis of neutrophil-to-lymphocyte and platelet-to-lymphocyte ratio in rheumatoid arthritis
Gian Luca Erre,Panagiotis Paliogiannis,Floriana Castagna,Arduino A. Mangoni,Ciriaco Carru,Giuseppe Passiu,Angelo Zinellu +6 more
TL;DR: A meta‐analysis is conducted to review the available evidence regarding the associations between peripheral blood neutrophil‐to‐lymphocyte ratio (NLR) and platelet‐to-lymphocytes ratio (PLR), and the presence of rheumatoid arthritis (RA).
Journal ArticleDOI
Outcome following isolated tricuspid valve replacement.
TL;DR: Isolated tricuspid valve replacement is characterized by a poor short and long-term outcome, with the median survival only 1.2 years and anasarca was the only predictor of short-term mortality.
Journal ArticleDOI
Temporal trends in anticholinergic medication prescription in older people: repeated cross-sectional analysis of population prescribing data
TL;DR: Despite increasing evidence of adverse outcomes, the proportion of older people prescribed anticholinergic medications and the proportion with a high anticholineergic exposure has increased between 1995 and 2010.
Journal ArticleDOI
Defining the COX inhibitor selectivity of NSAIDs: implications for understanding toxicity.
TL;DR: Data derived from in vitro models used to generate NSAID IC50 values informed the ‘imbalance hypothesis’ that the cardiovascular risk of NSAIDs results from an imbalance in the detrimental actions of COX-1-derived thromboxane A2 and the beneficial actions ofCOX-2-derived prostacyclin (PGI2).
Journal ArticleDOI
Impaired radial artery compliance in normotensive subjects with familial hypercholesterolemia
Cristina Giannattasio,Arduino A. Mangoni,Monica Failla,Stefano Carugo,M. L. Stella,Patrizia Stefanoni,Guido Grassi,Carlo Vergani,Giuseppe Mancia +8 more
TL;DR: In subjects with a marked increase in serum cholesterol due to FHC, not only arteriolar dilatation, but also RAC and distensibility are markedly impaired, and this impairment can be favourably affected by an effective lipid lowering treatment of long duration.