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Armando Aranda-Anzaldo

Researcher at Universidad Autónoma del Estado de México

Publications -  52
Citations -  633

Armando Aranda-Anzaldo is an academic researcher from Universidad Autónoma del Estado de México. The author has contributed to research in topics: Nuclear matrix & Virus. The author has an hindex of 16, co-authored 52 publications receiving 609 citations. Previous affiliations of Armando Aranda-Anzaldo include National Autonomous University of Mexico & Centre national de la recherche scientifique.

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NeuN/Fox-3 is an intrinsic component of the neuronal nuclear matrix.

TL;DR: Fox‐3 and Splicing factor SC35 colocalize by fluorescence microscopy and are repored as uniprotkb-Q6PDU1 and B7ZC13, respectively.
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Reassessing the role of p53 in cancer and ageing from an evolutionary perspective.

TL;DR: The evolutionary perspective indicates that p53 evolved so as to play a subtle but very important role during development while its role as a TSG is only important in animals that are protected from most sources of extrinsic mortality, thus suggesting that p 53 was primarily selected for its developmental role and not as aTSG.
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Chemical inactivation of human immunodeficiency virus in vitro.

TL;DR: The results suggest that HIV inactivation is dependent on the viral concentration, the time of incubation in presence of the putative disinfectant and the degree of virucidal activity of the latter.
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DNA moves sequentially towards the nuclear matrix during DNA replication in vivo.

TL;DR: Looped DNA moves in a sequential fashion, as if reeled in, towards the NM during DNA replication in vivo thus supporting the notion that the DNA template is pulled progressively towards the replication factories on the NM so as to be replicated.
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Gene positional changes relative to the nuclear substructure correlate with the proliferating status of hepatocytes during liver regeneration

TL;DR: Evidence is provided that transient changes in the topological relationships between specific genes and the nuclear substructure occur during liver regeneration and that such changes correlate with the actual proliferating status of the cells, suggesting that specific transitions in the higher-order DNA structure are characteristic of the quiescent (G0) and replicating (S) phases of the cell cycle in vivo.