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Arnold M. Moses
Researcher at State University of New York Upstate Medical University
Publications - 40
Citations - 1712
Arnold M. Moses is an academic researcher from State University of New York Upstate Medical University. The author has contributed to research in topics: Diabetes insipidus & Parathyroid hormone. The author has an hindex of 22, co-authored 40 publications receiving 1659 citations. Previous affiliations of Arnold M. Moses include Syracuse University & United States Department of Veterans Affairs.
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Journal ArticleDOI
Resistance to multiple hormones in patients with pseudohypoparathyroidism: Association with deficient activity of guanine nucleotide regulatory protein
Michael A. Levine,Robert W. Downs,Arnold M. Moses,Neil A. Breslau,Stephen J. Marx,Roz D. Lasker,Rene E. Rizzoli,Gerald D. Aurbach,Allen M. Spiegel +8 more
TL;DR: Researchers studied erythrocyte G unit activity and clinical endocrine function in 29 patients with pseudohypoparathyroidism type I to test the hypothesis that a defective G unit should lead to resistance to multiple hormones that act by stimulating adenylate cyclase.
Journal ArticleDOI
Bone quality assessment in type 2 diabetes mellitus.
TL;DR: Abnormal trabecular microarchitecture may help explain the paradox of increased fractures at a higher BMD in T2DM, and low TBS associates with worse glycemic control.
Journal ArticleDOI
Drug-induced dilutional hyponatremia.
Arnold M. Moses,Myron Miller +1 more
TL;DR: This report has been prepared to summarize the drugs and the problems that they have produced and to alert the medical profession to the problems.
Journal ArticleDOI
Pathophysiologic and pharmacologic alterations in the release and action of ADH.
Arnold M. Moses,Arnold M. Moses,Myron Miller,Myron Miller,David H.P. Streeten,David H.P. Streeten +5 more
TL;DR: The physiologic factors involved in vaseopressin (ADH) release and action are reviewed with emphasis on the interaction between osmotic and volume stimuli to the discharge of ADH.
Journal ArticleDOI
Identification and characterization of aquaporin-2 water channel mutations causing nephrogenic diabetes insipidus with partial vasopressin response
TL;DR: It is indicated that AQP2 mutations can confer partially responsive NDI, and co-injection of the mutant cRNAs with the wild-type cRNA did not affect the function of theWild-type AQP 2.