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Ashwini Chauhan

Researcher at Tripura University

Publications -  23
Citations -  1614

Ashwini Chauhan is an academic researcher from Tripura University. The author has contributed to research in topics: FtsZ & Biofilm. The author has an hindex of 16, co-authored 23 publications receiving 1379 citations. Previous affiliations of Ashwini Chauhan include Institute of Genomics and Integrative Biology & Pasteur Institute.

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From in vitro to in vivo Models of Bacterial Biofilm-Related Infections

TL;DR: A comprehensive analysis of the literature from a historic perspective commenting on the contribution of the different models and discussing future venues and new approaches that can be merged with more traditional techniques in order to model biofilm-infections and efficiently fight them is undertaken.
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Modulation of Mycobacterium tuberculosis proliferation by MtrA, an essential two-component response regulator.

TL;DR: It is proposed that proliferation of M.’tuberculosis in vivo depends, in part, on the optimal ratio of phosphorylated to non‐phosphorylated MtrA response regulator.
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Management of infections related to totally implantable venous-access ports: challenges and perspectives

TL;DR: In-vitro and in-vivo basic and clinical research findings on the epidemiology, diagnosis, and prevention of TIVAP-related infections, the current challenges to management, promising strategies, and some treatments in development that are likely to improve outcomes are discussed.
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Mycobacterium tuberculosis Cells Growing in Macrophages Are Filamentous and Deficient in FtsZ Rings

TL;DR: The results suggest that the intraphagosomal milieu alters the expression of M. tuberculosis genes affecting Z-ring formation and thereby cell division, and the levels of FtsZ in bacteria grown in macrophages or in broth were comparable, suggesting that Z- ring formation at midcell sites was compromised during intracellular growth.
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Interference of Mycobacterium tuberculosis cell division by Rv2719c, a cell wall hydrolase

TL;DR: It is proposed that Rv2719c is a potential regulator of M. tuberculosis cell division and that its levels, and possibly activities, are modulated under a variety of growth conditions including growth in vivo and during DNA damage, so that the assembly of FtsZ‐rings, and therefore the cell division, can proceed in a regulated manner.