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Barry J. Fetics

Researcher at Johns Hopkins University

Publications -  42
Citations -  6784

Barry J. Fetics is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Ejection fraction & Heart failure. The author has an hindex of 26, co-authored 42 publications receiving 6495 citations. Previous affiliations of Barry J. Fetics include Johns Hopkins University School of Medicine & Guidant.

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Journal ArticleDOI

Estimation of Central Aortic Pressure Waveform by Mathematical Transformation of Radial Tonometry Pressure Validation of Generalized Transfer Function

TL;DR: Central aortic pressures can be accurately estimated from radial tonometry with the use of a generalized TF, and the reconstructed waveform can provide arterial compliance estimates but may underestimate the augmentation index because the latter requires greater fidelity reproduction of the wave contour.
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Improved Left Ventricular Mechanics From Acute VDD Pacing in Patients With Dilated Cardiomyopathy and Ventricular Conduction Delay

TL;DR: VDD pacing acutely enhances contractile function in heart failure patients with intraventricular conduction delay and single-site pacing at the site of greatest delay achieves similar or greater benefits to biventricular pacing in such patients, to clarify pacing-effect mechanisms.
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Combined Ventricular Systolic and Arterial Stiffening in Patients With Heart Failure and Preserved Ejection Fraction. Implications for Systolic and Diastolic Reserve Limitations

TL;DR: Patients with HF-lnEF have systolic-ventricular and arterial stiffening beyond that associated with aging and/or hypertension, which may play an important pathophysiological role by exacerbating systemic load interaction with diastolic function, augmenting blood pressure lability, and elevating cardiac metabolic demand under stress.
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Left ventricular or biventricular pacing improves cardiac function at diminished energy cost in patients with dilated cardiomyopathy and left bundle-branch block

TL;DR: Ventricular resynchronization by left ventricular or biventricular pacing/stimulation in DCM patients with left bundle-branch block acutely enhances systolic function while modestly lowering energy cost should prove valuable for treating DCM Patients with basal dyssynchrony.
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Noninvasive single-beat determination of left ventricular end-systolic elastance in humans

TL;DR: The E(es) can be reliably estimated from simple noninvasive measurements and should broaden the clinical applicability of this useful parameter for assessing systolic function, therapeutic response and ventricular-arterial interaction.