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Betty Soliven
Researcher at University of Chicago
Publications - 90
Citations - 4407
Betty Soliven is an academic researcher from University of Chicago. The author has contributed to research in topics: Myasthenia gravis & Myelin. The author has an hindex of 36, co-authored 85 publications receiving 4026 citations. Previous affiliations of Betty Soliven include NewYork–Presbyterian Hospital & University of Illinois at Chicago.
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Phosphorylation of myelin basic protein in intact oligodendrocytes: inhibition by galactosylsphingosine and cyclic AMP.
TL;DR: A role for the interregulation of protein kinase A (PKA) and PKC in the control of OLG somal vs. myelin components is emphasized; this may have significant implications for central nervous system myelin assembly.
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Forskolin and phorbol esters decrease the same K+ conductance in cultured oligodendrocytes.
TL;DR: It is postulated that the potassium currents in OLG can be physiologically modulated by two distinct second-messenger systems, perhaps converging at the level of a common phosphorylated enzyme or regulatory protein.
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Myasthenia gravis, an autoimmune manifestation of lymphoma and lymphoproliferative disorders: case reports and review of literature
TL;DR: The pathogenesis of MG in lymphoid malignancies is probably heterogeneous and likely relates to perturbations in the immune mechanisms that normally prevent the emergence of autoimmunity.
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Chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) with hypertrophic spinal radiculopathy mimicking neurofibromatosis.
TL;DR: An autopsy 15 years after the onset of symptoms revealed hypertrophic radiculopathy and peripheral neuropathy due to CIDP with no evidence of neurofibromatosis, illustrating how the hypertrophic neuropathy accompanying C IDP can be mistaken for neurof fibroma.
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Evaluation of neuropathy in patients on suramin treatment
Betty Soliven,Upinder K. Dhand,Ken Kobayashi,Ravinder Arora,Barbara Martin,Michele V. Petersen,Linda Janisch,Nicholas J. Vogelzang,Everett E. Vokes,Mark J. Ratain +9 more
TL;DR: It is concluded that serial electrophysiologic monitoring is helpful for early detection of suramin‐induced neuropathy and fixed dosing schedule of suramine without adaptive control does not lead to an increased incidence of demyelinating neuropathy when compared to adaptively controlled dosing schedules.