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Bo Shen
Researcher at Guangzhou Medical University
Publications - 5
Citations - 287
Bo Shen is an academic researcher from Guangzhou Medical University. The author has contributed to research in topics: Cell & Oncogene. The author has an hindex of 5, co-authored 5 publications receiving 240 citations.
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Journal ArticleDOI
Association of Fusobacterium nucleatum infection with colorectal cancer in Chinese patients
Yu Yuan Li,Quan Xing Ge,Jie Cao,Yong Jian Zhou,Yanlei Du,Bo Shen,Yu-Jui Yvonne Wan,Yu Qiang Nie +7 more
TL;DR: F. nucleatum was enriched in CRC tissues and associated with CRC development and metastasis and its association with CRC invasiveness in Chinese patients was observed.
Journal ArticleDOI
MicroRNA-181a promotes gastric cancer by negatively regulating tumor suppressor KLF6
TL;DR: It is found that miR-181a is overexpressed in human gastric cancer tissues and functions as an oncomir in Gastric cancer by repressing the expression of tumor suppressor KLF6.
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Genetic polymorphism at miR-181a binding site contributes to gastric cancer susceptibility
Yong Lin,Yuqiang Nie,Jing Zhao,Xi Chen,Min Ye,Yingfei Li,Yanlei Du,Jie Cao,Bo Shen,Yuyuan Li +9 more
TL;DR: It is suggested that rs12537 is associated with susceptibility and prognosis of GC in southern Han Chinese, and miR-181a and its target gene MTMR3 play important roles in GC.
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Expression and inhibitory role of TIMP-3 in hepatocellular carcinoma
Bo Shen,Yingjie Jiang,Yuan-Ran Chen,Hui-Cong Zheng,Wei Zeng,Yuyuan Li,Aoxian Yin,Yuqiang Nie +7 more
TL;DR: TIMP-3 expression is suppressed by promoter methylation in HCC, which acts as a functional tumor suppressor by inhibiting HCC cell proliferation, invasion, and migration and by inducing apoptosis and cell cycle arrest at the G2/M phase.
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NIMA-related kinase 2 regulates hepatocellular carcinoma cell growth and proliferation.
Xiao‑Bo Lai,Yu Qiang Nie,Hong‑Li Huang,Ying‑Fei Li,Chuang‑Yu Cao,Hui Yang,Bo Shen,Zhi‑Qiang Feng +7 more
TL;DR: Inhibiting Nek2 expression by siRNA suppressed cell proliferation, growth, and colony formation in hepatocellular carcinoma cell line HepG2 cells, and demonstrates that overexpression of Nek2 is associated with the malignant evolution of HCC.