Brian A. Ellison

TL;DR: It is demonstrated that a paucity of galanin-immunoreactive intermediate nucleus neurons is accompanied by sleep fragmentation in older adults with and without Alzheimer's disease.

TL;DR: An approach that accounts for changes in velocity during walking and allows for translation across species is presented, suitable to quantify qualitative walking abnormalities related to CNS circuit dysfunction across species, identify appropriate animal models, and it provides important translational opportunities.

TL;DR: The relative position of spinal motoneuron pools is conserved across species, including primates, and one can assign spinal cord levels to even single sections by matching ventral horn shape to standardized series in humans.

TL;DR: A normal number of galanin-immunoreactive VLPO neurons in narcolepsy type 1 brains at autopsy suggests that VL PO cell loss is an unlikely explanation for the sleep fragmentation that often accompanies the disease.

TL;DR: It is hypothesized that quantitative susceptibility mapping (QSM) MRI, which maps tissue susceptibility, demonstrates less in- and through-plane intensity variation, improving the clinician's ability to categorize a finding as a CMB.