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Bruce C. Nisula
Researcher at National Institutes of Health
Publications - 103
Citations - 7161
Bruce C. Nisula is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Human chorionic gonadotropin & Sialic acid. The author has an hindex of 37, co-authored 103 publications receiving 6833 citations. Previous affiliations of Bruce C. Nisula include University of New Mexico & French Institute of Health and Medical Research.
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Journal ArticleDOI
Incidence of Early Loss of Pregnancy
Allen J. Wilcox,Clarice R. Weinberg,John O'Connor,Donna D. Baird,John Schlatterer,Robert E. Canfield,E G Armstrong,Bruce C. Nisula +7 more
TL;DR: The total rate of pregnancy loss after implantation, including clinically recognized spontaneous abortions, was 31 percent and most of the 40 women with unrecognized early pregnancy losses had normal fertility, since 95 percent of them subsequently became clinically pregnant within two years.
Journal ArticleDOI
Transport of Steroid Hormones: Binding of 21 Endogenous Steroids to Both Testosterone-Binding Globulin and Corticosteroid-Binding Globulin in Human Plasma
TL;DR: A model of steroid transport in human plasma is described, finding that normal men, normal women during both the follicular and luteal phases of the ovarian cycle, and women during the third trimester of a normal pregnancy are candidates for steroid transport.
Journal ArticleDOI
Successful treatment of Cushing's syndrome with the glucocorticoid antagonist RU 486.
Lynnette K. Nieman,George P. Chrousos,Charles H. Kellner,Irving M. Spitz,Bruce C. Nisula,Gordon B. Cutler,George R. Merriam,C. Wayne Bardin,D. Lynn Loriaux +8 more
TL;DR: It is concluded that RU 486 may provide a safe, well tolerated, and effective medical treatment for hypercortisolism.
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Transport of Steroid Hormones: Interaction of 70 Drugs with Testosterone-Binding Globulin and Corticosteroid-Binding Globulin in Human Plasma
TL;DR: The results indicate that binding to steroid transport proteins should be considered among the in vivo effects of drugs on endogenous steroid hormone levels.
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A potential novel mechanism for precocious puberty in juvenile hypothyroidism.
TL;DR: Rec-hTSH acted as a competitive inhibitor of hF SH at the hFSH-R, indicating that hTSH and hFsh are acting through the same receptor, namely the hHTSH, and provides a potential novel mechanism for the precocious puberty of juvenile hypothyroidism.