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Byung Soo Kim

Researcher at Korea University

Publications -  141
Citations -  2929

Byung Soo Kim is an academic researcher from Korea University. The author has contributed to research in topics: Induced pluripotent stem cell & Multiple myeloma. The author has an hindex of 26, co-authored 141 publications receiving 2367 citations. Previous affiliations of Byung Soo Kim include Korea University Medical Center & ETH Zurich.

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Deep Fluids: A Generative Network for Parameterized Fluid Simulations

TL;DR: In this paper, a convolutional neural network is trained on a collection of discrete, parameterizable fluid simulation velocity fields to synthesize fluid simulations from a set of reduced parameters.
Journal ArticleDOI

Deep Fluids: A Generative Network for Parameterized Fluid Simulations

TL;DR: In this article, a convolutional neural network is trained on a collection of discrete, parameterizable fluid simulation velocity fields to synthesize fluid simulations from a set of reduced parameters.
Journal Article

Sinus augmentation utilizing anorganic bovine bone (Bio-Oss) with absorbable and nonabsorbable membranes placed over the lateral window: histomorphometric and clinical analyses.

TL;DR: There was no significant difference between the membrane groups as to vital bone formation and implant survival and the results of sinus elevation performed without a membrane were compared with either a short-term bioabsorbable membrane (Bio-Gide) or a nonabsorbables membrane (Gore-Tex).
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Action potential duration restitution kinetics in human atrial fibrillation

TL;DR: Atrial fibrillation was related to steeply sloped APDR kinetics and the spatial dispersion of APDR in patients with chronic AF was greater than that of patients with paroxysmal AF and control subjects, indicating that the heterogeneity ofAPDR of the atrium plays an important role in the persistence of AF.
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Drug resistance to 5-FU linked to reactive oxygen species modulator 1.

TL;DR: It is shown that enforced Romo1 expression induces reactive oxygen species (ROS) production in the mitochondria leading to massive cell death, and cellular adaptive response to Romo1-induced ROS is another mechanism of drug resistance to 5-FU.