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Carter A. Mitchell

Researcher at National Institutes of Health

Publications -  15
Citations -  2042

Carter A. Mitchell is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Aldosterone & Mineralocorticoid. The author has an hindex of 10, co-authored 15 publications receiving 1914 citations. Previous affiliations of Carter A. Mitchell include Woodward, Inc. & West Virginia University.

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Aldosterone-mediated regulation of ENaC α, β, and γ subunit proteins in rat kidney

TL;DR: At the protein level, the response of ENaC to aldosterone stimulation is heterogenous, with both quantitative and qualitative changes that can explain observed increases in ENac-mediated sodium transport.
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The thiazide-sensitive Na–Cl cotransporter is an aldosterone-induced protein

TL;DR: It is concluded that the distal convoluted tubule is an important site of action of the mineralocorticoid aldosterone, which strongly up-regulates the expression of TSC.
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Vasopressin increases Na-K-2Cl cotransporter expression in thick ascending limb of Henle’s loop

TL;DR: It is concluded that vasopressin strongly upregulates the expression of the Na-K-2Cl cotransporter of the TAL and that it is likely to play an important role in the long-term regulation of the countercurrent multiplication system.
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Vasopressin-mediated regulation of epithelial sodium channel abundance in rat kidney

TL;DR: All three subunits of ENaC are upregulated by vasopressin with temporal and regional differences, but changes are too slow to play a major role in the short-term action of vasoppressin to stimulate sodium reabsorption in the collecting duct.
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UT-A2: a 55-kDa urea transporter in thin descending limb whose abundance is regulated by vasopressin.

TL;DR: Results indicate that UT-A2 is expressed as a 55-kDa protein in portions of the thin descending limbs of Henle's loop and that the abundance of this protein is strongly upregulated by vasopressin.