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Cassandra A. Lee

Researcher at University of California, Davis

Publications -  44
Citations -  2300

Cassandra A. Lee is an academic researcher from University of California, Davis. The author has contributed to research in topics: Anterior cruciate ligament & Cartilage. The author has an hindex of 21, co-authored 39 publications receiving 1860 citations. Previous affiliations of Cassandra A. Lee include Boston Medical Center & Wake Forest Baptist Medical Center.

Papers
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Journal ArticleDOI

Surgical and tissue engineering strategies for articular cartilage and meniscus repair.

TL;DR: Surgical techniques, including tissue-engineered products, that are currently in clinical use, as well as a discussion of state-of-the-art tissue engineering strategies and technologies that are being developed for use in articular cartilage and meniscus repair and regeneration are provided.
Journal ArticleDOI

Analysis of Outcomes of Anterior Cruciate Ligament Repair With 5-Year Follow-up: Allograft Versus Autograft

TL;DR: Overall, the allograft patients reported less pain at 1 and 6 weeks after surgery, better function at 1 week, 3 months, and 1 year, and fewer activity limitations throughout the follow-up period.
Book ChapterDOI

The Bone Organ System: Form and Function

TL;DR: The information and concepts described in this chapter form the basis for understanding bone as a tissue and as an organ system and more information is needed before a truly comprehensive understanding of the bone organ system can be developed.
Journal ArticleDOI

Expression of angiogenic factors during distraction osteogenesis

TL;DR: It is suggested that bone formation during distraction osteogenesis is accompanied by the robust induction of factors associated with angiogenesis and support further investigations to elucidate the mechanisms by which angiogenic events promote bone repair and regeneration.
Journal ArticleDOI

Musculoskeletal changes following non-invasive knee injury using a novel mouse model of post-traumatic osteoarthritis

TL;DR: A novel mouse model of PTOA that injures the ACL with a single load of tibial compression overload is established, and the time course of musculoskeletal changes following knee injury is described, helping to establish the window of opportunity for preventative treatment.