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Chan-Min Liu

Researcher at Jiangsu Normal University

Publications -  40
Citations -  2541

Chan-Min Liu is an academic researcher from Jiangsu Normal University. The author has contributed to research in topics: Oxidative stress & Lipid peroxidation. The author has an hindex of 26, co-authored 37 publications receiving 2122 citations.

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Quercetin activates AMP-activated protein kinase by reducing PP2C expression protecting old mouse brain against high cholesterol-induced neurotoxicity

TL;DR: Oral administration of quercetin significantly improved the behavioural performance of high‐cholesterol‐fed old mice in both a step‐through test and the Morris water maze task, and suggest that AMPK activated by quercETin may be a potential target to enhance the resistance of neurons to age‐related diseases.
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Protective role of quercetin against lead-induced inflammatory response in rat kidney through the ROS-mediated MAPKs and NF-κB pathway

TL;DR: The inhibition of Pb-induced kidney inflammation by quercetin is due at least in part to its anti-oxidant activity and its ability to modulate the MAPK and NF-κB signaling pathway.
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Ursolic Acid Attenuates D-Galactose-Induced Inflammatory Response in Mouse Prefrontal Cortex through Inhibiting AGEs/RAGE/NF-κB Pathway Activation

TL;DR: Ursolic acid administration significantly improved behavioral performance of D-gal-treated mice in step-through test and Morris water maze task and significantly decreased AGEs induced the expression of receptor for advanced glycation end products and inhibited NF-κB p65 nuclear translocation in the prefrontal cortex of D -gal- treated mice.
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Quercetin protects rat liver against lead-induced oxidative stress and apoptosis

TL;DR: The data suggest that quercetin protects the rat liver from lead-induced injury by attenuating lipid peroxidation, renewing the activities of antioxidant enzymes and inhibiting apoptosis.
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Puerarin protects rat kidney from lead-induced apoptosis by modulating the PI3K/Akt/eNOS pathway

TL;DR: Results suggested that the inhibition of Pb-induced apoptosis by puerarin is due at least in part to its antioxidant activity and its ability to modulate the PI3K/Akt/eNOS signaling pathway.