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Chaoying Qin

Researcher at Central South University

Publications -  20
Citations -  179

Chaoying Qin is an academic researcher from Central South University. The author has contributed to research in topics: Medicine & Immunotherapy. The author has an hindex of 5, co-authored 11 publications receiving 64 citations.

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circCPA4 acts as a prognostic factor and regulates the proliferation and metastasis of glioma.

TL;DR: It is found that circCPA4 (hsa_circ_0082374) up‐regulated the most in glioma tissues and high levels of circCpa4 were positively related to poor outcome of gliomas, and circC PA4 could be a novel prognostic biomarker and target for gli cancer treatment.
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Identification and validation of a three-gene signature as a candidate prognostic biomarker for lower grade glioma.

TL;DR: This study identified a three-gene model that showed satisfactory performance in predicting the 1, 3- and 5-year survival of LGG patients compared to other models and may be a promising independent biomarker of L GG.
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Interplay of m6A and histone modifications contributes to temozolomide resistance in glioblastoma

TL;DR: Zhang et al. as mentioned in this paper demonstrated that TMZ treatment upregulated the expression of the m6 A methyltransferase METTL3, thereby increasing the modification of histone modification-related gene transcripts.
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Overexpression of the phospholipase A2 group V gene in glioma tumors is associated with poor patient prognosis.

TL;DR: It is found that high PLA2G5 gene expression was associated with unfavorable prognosis in both low-grade and high-grade gliomas, and indicates that PLA1G5 could be a potential biomarker for predicting poor prog outlook in patients with glioma.
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LINC00470 promotes tumour proliferation and invasion, and attenuates chemosensitivity through the LINC00470/miR-134/Myc/ABCC1 axis in glioma.

TL;DR: It is concluded that LINC00470 promoted the expression of MYC and ABCC1 by suppressing miR‐134, thus promoting glioma cell proliferation and invasion, and attenuating TMZ chemosensitivity, and the Linc00470/miR‐ 134/MYC/ABCC1 axis constitutes a potential therapeutic target.